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Nature Communications|November 12, 2021
Acute myeloid leukemia maturation lineage influences residual disease and relapse following differentiation therapySteven Ngo, Ethan P Oxley, Margherita Ghisi, et al.Nature Communications|May 6, 2025
Spatial and stoichiometric in situ analysis of biomolecular oligomerization at single-protein resolutionLuciano A Masullo, Rafal Kowalewski, Monique Honsa, et al.Cell Reports|October 9, 2024
Context-restricted PD-(L)1 checkpoint agonism by CTLA4-Ig therapies inhibits T cell activityEthan P Oxley, Nadia J Kershaw, Cynthia Louis, et al.Nature Communications|July 7, 2021
Oncogenic cooperation between TCF7-SPI1 and NRAS(G12D) requires β-catenin activity to drive T-cell acute lymphoblastic leukemiaQuentin Van Thillo, Jolien De Bie, Janith A Seneviratne, et al.Cell Stem Cell|August 3, 2019
Interconversion between Tumorigenic and Differentiated States in Acute Myeloid LeukemiaMark D McKenzie, Margherita Ghisi, Ethan P Oxley, et al.Blood|May 6, 2020
The EMT modulator SNAI1 contributes to AML pathogenesis via its interaction with LSD1Catherine L Carmichael, Jueqiong Wang, Thao Nguyen, et al.Pageof 1