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Nature Reviews. Molecular Cell Biology|September 16, 2010
Pathways of mammalian replication fork restartEva Petermann, Thomas HelledayProceedings of the National Academy of Sciences of the United States of America|September 1, 2010
Chk1 promotes replication fork progression by controlling replication initiationEva Petermann, Mick Woodcock, Thomas HelledayMolecular Biology of the Cell|March 21, 2008
Claspin promotes normal replication fork rates in human cellsEva Petermann, Thomas Helleday, Keith W CaldecottProceedings of the National Academy of Sciences of the United States of America|December 19, 2008
Essential function of Chk1 can be uncoupled from DNA damage checkpoint and replication controlDeborah Wilsker, Eva Petermann, Thomas Helleday, et al.Nature Reviews. Cancer|February 8, 2008
DNA repair pathways as targets for cancer therapyThomas Helleday, Eva Petermann, Cecilia Lundin, et al.Molecular Cell|March 2, 2010
Hydroxyurea-stalled replication forks become progressively inactivated and require two different RAD51-mediated pathways for restart and repairEva Petermann, Manuel Luís Orta, Natalia Issaeva, et al.Molecular Cancer Therapeutics|November 16, 2011
Targeted mutations in the ATR pathway define agent-specific requirements for cancer cell growth and survivalDeborah Wilsker, Jon H Chung, Ivan Pradilla, et al.Journal of Molecular Biology|July 21, 2010
Methylated DNA causes a physical block to replication forks independently of damage signalling, O(6)-methylguanine or DNA single-strand breaks and results in DNA damagePetra Groth, Simon Ausländer, Muntasir Mamun Majumder, et al.Cancer Treatment and Research|November 17, 2023
Mitotic MTH1 Inhibitors in Treatment of CancerThomas HelledayEuropean Journal of Cancer (Oxford, England : 1990)|April 1, 2008
Amplifying tumour-specific replication lesions by DNA repair inhibitors - a new era in targeted cancer therapyThomas HelledayPageof 30