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Eva R Chin

Showing results (11-20 of 31) with videos related to

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American Journal of Physiology. Cell Physiology|September 26, 2014
Perturbations in intracellular Ca2+ handling in skeletal muscle in the G93A*SOD1 mouse model of amyotrophic lateral sclerosisEva R Chin, Dapeng Chen, Kostyantyn D Bobyk, et al.
Human Molecular Genetics|March 23, 2006
Targeted inhibition of Ca2+ /calmodulin signaling exacerbates the dystrophic phenotype in mdx mouse muscleJoe V Chakkalakal, Stephanie A Michel, Eva R Chin, et al.
The Journals of Gerontology. Series A, Biological Sciences and Medical Sciences|January 15, 2014
Telomeres shorten in response to oxidative stress in mouse skeletal muscle fibersAndrew T Ludlow, Espen E Spangenburg, Eva R Chin, et al.
Applied Physiology, Nutrition, and Metabolism = Physiologie Appliquee, Nutrition Et Metabolisme|December 7, 2007
Ca2+/calmodulin-based signalling in the regulation of the muscle fibre phenotype and its therapeutic potential via modulation of utrophin A and myostatin expressionRobin N Michel, Eva R Chin, Joe V Chakkalakal, et al.
Skeletal Muscle|April 14, 2016
The SH3 and cysteine-rich domain 3 (Stac3) gene is important to growth, fiber composition, and calcium release from the sarcoplasmic reticulum in postnatal skeletal muscleXiaofei Cong, Jonathan Doering, Davi A G Mazala, et al.
American Journal of Physiology. Cell Physiology|February 6, 2015
SERCA1 overexpression minimizes skeletal muscle damage in dystrophic mouse modelsDavi A G Mázala, Stephen J P Pratt, Dapeng Chen, et al.
Diabetes|February 25, 2016
Response to Comment on Prior et al. Increased Skeletal Muscle Capillarization Independently Enhances Insulin Sensitivity in Older Adults After Exercise Training and Detraining. Diabetes 2015;64:3386-3395Steven J Prior, Andrew P Goldberg, Heidi K Ortmeyer, et al.
International Journal of Toxicology|March 20, 2025
A Characterization of the Nonclinical Pharmacology and Toxicology of Aficamten, a Reversible Allosteric Inhibitor of Cardiac MyosinMichael K Pugsley, Darren T Hwee, Brett R Winters, et al.
The Biochemical Journal|March 20, 2004
Mechanical stimuli regulate rapamycin-sensitive signalling by a phosphoinositide 3-kinase-, protein kinase B- and growth factor-independent mechanismTroy A Hornberger, Rudy Stuppard, Kevin E Conley, et al.
Diabetes|June 13, 2015
Increased Skeletal Muscle Capillarization Independently Enhances Insulin Sensitivity in Older Adults After Exercise Training and DetrainingSteven J Prior, Andrew P Goldberg, Heidi K Ortmeyer, et al.
Pageof 4

Showing results (11-20 of 31) with videos related to

Sort By:
Pageof 4
American Journal of Physiology. Cell Physiology|September 26, 2014
Perturbations in intracellular Ca2+ handling in skeletal muscle in the G93A*SOD1 mouse model of amyotrophic lateral sclerosisEva R Chin, Dapeng Chen, Kostyantyn D Bobyk, et al.
Human Molecular Genetics|March 23, 2006
Targeted inhibition of Ca2+ /calmodulin signaling exacerbates the dystrophic phenotype in mdx mouse muscleJoe V Chakkalakal, Stephanie A Michel, Eva R Chin, et al.
The Journals of Gerontology. Series A, Biological Sciences and Medical Sciences|January 15, 2014
Telomeres shorten in response to oxidative stress in mouse skeletal muscle fibersAndrew T Ludlow, Espen E Spangenburg, Eva R Chin, et al.
Applied Physiology, Nutrition, and Metabolism = Physiologie Appliquee, Nutrition Et Metabolisme|December 7, 2007
Ca2+/calmodulin-based signalling in the regulation of the muscle fibre phenotype and its therapeutic potential via modulation of utrophin A and myostatin expressionRobin N Michel, Eva R Chin, Joe V Chakkalakal, et al.
Skeletal Muscle|April 14, 2016
The SH3 and cysteine-rich domain 3 (Stac3) gene is important to growth, fiber composition, and calcium release from the sarcoplasmic reticulum in postnatal skeletal muscleXiaofei Cong, Jonathan Doering, Davi A G Mazala, et al.
American Journal of Physiology. Cell Physiology|February 6, 2015
SERCA1 overexpression minimizes skeletal muscle damage in dystrophic mouse modelsDavi A G Mázala, Stephen J P Pratt, Dapeng Chen, et al.
Diabetes|February 25, 2016
Response to Comment on Prior et al. Increased Skeletal Muscle Capillarization Independently Enhances Insulin Sensitivity in Older Adults After Exercise Training and Detraining. Diabetes 2015;64:3386-3395Steven J Prior, Andrew P Goldberg, Heidi K Ortmeyer, et al.
International Journal of Toxicology|March 20, 2025
A Characterization of the Nonclinical Pharmacology and Toxicology of Aficamten, a Reversible Allosteric Inhibitor of Cardiac MyosinMichael K Pugsley, Darren T Hwee, Brett R Winters, et al.
The Biochemical Journal|March 20, 2004
Mechanical stimuli regulate rapamycin-sensitive signalling by a phosphoinositide 3-kinase-, protein kinase B- and growth factor-independent mechanismTroy A Hornberger, Rudy Stuppard, Kevin E Conley, et al.
Diabetes|June 13, 2015
Increased Skeletal Muscle Capillarization Independently Enhances Insulin Sensitivity in Older Adults After Exercise Training and DetrainingSteven J Prior, Andrew P Goldberg, Heidi K Ortmeyer, et al.
Pageof 4