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Journal of Molecular and Cellular Cardiology|December 31, 2013
Genetic variation in the two-pore domain potassium channel, TASK-1, may contribute to an atrial substrate for arrhythmogenesisBo Liang, Magdalena Soka, Alex Horby Christensen, et al.The Journal of Clinical Investigation|May 3, 2008
GLUT1 mutations are a cause of paroxysmal exertion-induced dyskinesias and induce hemolytic anemia by a cation leakYvonne G Weber, Alexander Storch, Thomas V Wuttke, et al.Molecular Psychiatry|February 25, 2020
Multiple inducers and novel roles of autoantibodies against the obligatory NMDAR subunit NR1: a translational study from chronic life stress to brain injuryHong Pan, Agnes A Steixner-Kumar, Anna Seelbach, et al.Annals of Clinical and Translational Neurology|November 22, 2017
NMDAR encephalitis: passive transfer from man to mouse by a recombinant antibodyManish Malviya, Sumanta Barman, Kristin S Golombeck, et al.Molecular Psychiatry|February 28, 2024
GRID1/GluD1 homozygous variants linked to intellectual disability and spastic paraplegia impair mGlu1/5 receptor signaling and excitatory synapsesDévina C Ung, Nicolas Pietrancosta, Elena Baz Badillo, et al.Cell Research|October 27, 2021
K<sub>2P</sub>18.1 translates T cell receptor signals into thymic regulatory T cell developmentTobias Ruck, Stefanie Bock, Steffen Pfeuffer, et al.Molecular Psychiatry|February 11, 2018
Uncoupling the widespread occurrence of anti-NMDAR1 autoantibodies from neuropsychiatric disease in a novel autoimmune modelHong Pan, Bárbara Oliveira, Gesine Saher, et al.The Journal of Clinical Investigation|January 31, 2023
Neuron-oligodendrocyte potassium shuttling at nodes of Ranvier protects against inflammatory demyelinationHannah Kapell, Luca Fazio, Julia Dyckow, et al.Pageof 20