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Pflugers Archiv : European Journal of Physiology
|
January 21, 2018
Angiotensin-(1-7)-induced Mas receptor activation attenuates atherosclerosis through a nitric oxide-dependent mechanism in apolipoproteinE-KO mice
Guang Yang, Geoffrey Istas, Sascha Höges, et al.
Free Radical Biology & Medicine
|
October 18, 2015
Left ventricular diastolic dysfunction in Nrf2 knock out mice is associated with cardiac hypertrophy, decreased expression of SERCA2a, and preserved endothelial function
Ralf Erkens, Christian M Kramer, Wiebke Lückstädt, et al.
The American Journal of Pathology
|
June 19, 2016
Proatherosclerotic Effect of the α1-Subunit of Soluble Guanylyl Cyclase by Promoting Smooth Muscle Phenotypic Switching
Maria Segura-Puimedon, Evanthia Mergia, Jaafar Al-Hasani, et al.
Circulation Research
|
December 4, 2004
cGMP catabolism by phosphodiesterase 5A regulates cardiac adrenergic stimulation by NOS3-dependent mechanism
Eiki Takimoto, Hunter C Champion, Diego Belardi, et al.
Redox Biology
|
October 13, 2017
Identification of a soluble guanylate cyclase in RBCs: preserved activity in patients with coronary artery disease
Miriam M Cortese-Krott, Evanthia Mergia, Christian M Kramer, et al.
Molecular Pharmacology
|
September 7, 2017
NO-Sensitive Guanylate Cyclase Isoforms NO-GC1 and NO-GC2 Contribute to Noise-Induced Inner Hair Cell Synaptopathy
Dorit Möhrle, Katrin Reimann, Steffen Wolter, et al.
The Journal of Clinical Investigation
|
September 1, 2023
Hypoxic erythrocytes mediate cardioprotection through activation of soluble guanylate cyclase and release of cyclic GMP
Jiangning Yang, Michaela L Sundqvist, Xiaowei Zheng, et al.
Cardiovascular Research
|
June 23, 2026
Carbon dioxide is a triple vasodilator
Dragos A Duse, Nathalie H Schröder, Sotirios Akritidis, et al.
Nature
|
November 12, 2013
Dysfunctional nitric oxide signalling increases risk of myocardial infarction
Jeanette Erdmann, Klaus Stark, Ulrike B Esslinger, et al.
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of 4
Search research articles
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Showing results (31-40 of 39) with videos related to
Sort By:
Page
of 4
You have reached the last page of results.
This site can display upto 39 results.
Pflugers Archiv : European Journal of Physiology
|
January 21, 2018
Angiotensin-(1-7)-induced Mas receptor activation attenuates atherosclerosis through a nitric oxide-dependent mechanism in apolipoproteinE-KO mice
Guang Yang, Geoffrey Istas, Sascha Höges, et al.
Free Radical Biology & Medicine
|
October 18, 2015
Left ventricular diastolic dysfunction in Nrf2 knock out mice is associated with cardiac hypertrophy, decreased expression of SERCA2a, and preserved endothelial function
Ralf Erkens, Christian M Kramer, Wiebke Lückstädt, et al.
The American Journal of Pathology
|
June 19, 2016
Proatherosclerotic Effect of the α1-Subunit of Soluble Guanylyl Cyclase by Promoting Smooth Muscle Phenotypic Switching
Maria Segura-Puimedon, Evanthia Mergia, Jaafar Al-Hasani, et al.
Circulation Research
|
December 4, 2004
cGMP catabolism by phosphodiesterase 5A regulates cardiac adrenergic stimulation by NOS3-dependent mechanism
Eiki Takimoto, Hunter C Champion, Diego Belardi, et al.
Redox Biology
|
October 13, 2017
Identification of a soluble guanylate cyclase in RBCs: preserved activity in patients with coronary artery disease
Miriam M Cortese-Krott, Evanthia Mergia, Christian M Kramer, et al.
Molecular Pharmacology
|
September 7, 2017
NO-Sensitive Guanylate Cyclase Isoforms NO-GC1 and NO-GC2 Contribute to Noise-Induced Inner Hair Cell Synaptopathy
Dorit Möhrle, Katrin Reimann, Steffen Wolter, et al.
The Journal of Clinical Investigation
|
September 1, 2023
Hypoxic erythrocytes mediate cardioprotection through activation of soluble guanylate cyclase and release of cyclic GMP
Jiangning Yang, Michaela L Sundqvist, Xiaowei Zheng, et al.
Cardiovascular Research
|
June 23, 2026
Carbon dioxide is a triple vasodilator
Dragos A Duse, Nathalie H Schröder, Sotirios Akritidis, et al.
Nature
|
November 12, 2013
Dysfunctional nitric oxide signalling increases risk of myocardial infarction
Jeanette Erdmann, Klaus Stark, Ulrike B Esslinger, et al.
Page
of 4