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The Journal of Pharmacology and Experimental Therapeutics|November 1, 1996
Pharmacological properties of acquired excitotoxicity in Chinese hamster ovary cells transfected with N-methyl-D-aspartate receptor subunitsF A Boeckman, E AizenmanNeuroscience Letters|May 23, 1994
Stable transfection of the NR1 subunit in Chinese hamster ovary cells fails to produce a functional N-methyl-D-aspartate receptorF A Boeckman, E AizenmanProceedings of the National Academy of Sciences of the United States of America|October 6, 1997
Functional consequences of NR2 subunit composition in single recombinant N-methyl-D-aspartate receptorsJ C Brimecombe, F A Boeckman, E AizenmanBrain Research|February 7, 1997
Intrinsic redox properties of N-methyl-D-aspartate receptor can determine the developmental expression of excitotoxicity in rat cortical neurons in vitroJ D Sinor, F A Boeckman, E AizenmanNeuroscience Letters|September 14, 1992
Glutathione prevents 2,4,5-trihydroxyphenylalanine excitotoxicity by maintaining it in a reduced, non-active formE Aizenman, F A Boeckman, P A RosenbergOncogene|July 1, 1991
N-myc oncogene enhances mitogenic responsiveness of diploid human fibroblasts to growth factors but fails to immortalizeW H Brondyk, F A Boeckman, W E FahlJournal of Neurophysiology|January 1, 1997
Trapping channel block of NMDA-activated responses by amantadine and memantineT A Blanpied, F A Boeckman, E Aizenman, et al.Biochemistry|January 20, 1999
Solution structure of the sodium channel inactivation gateC A Rohl, F A Boeckman, C Baker, et al.Pageof 1