Showing results (1-10 of 515) with videos related to
Sort By:
Pageof 52
Lancet (London, England)|August 28, 1999
Lactate is an unreliable indicator of tissue hypoxia in injury or sepsisJ H James, F A Luchette, F D McCarter, et al.Pharmacology|January 1, 1981
Transport of neutral amino acids at the blood-brain barrierJ H James, J E FischerThe Journal of Surgical Research|July 27, 2001
Adrenergic blockade reduces skeletal muscle glycolysis and Na(+), K(+)-ATPase activity during hemorrhageF D McCarter, J H James, F A Luchette, et al.The Journal of Trauma|May 29, 1998
Increased skeletal muscle Na+, K+-ATPase activity as a cause of increased lactate production after hemorrhagic shockF A Luchette, L A Friend, C C Brown, et al.Science (New York, N.Y.)|June 23, 1978
Blood-brain neutral amino acid transport activity is increased after portacaval anastomosisJ H James, J Escourrou, J E FischerThe Journal of Clinical Investigation|January 15, 1997
Sepsis is associated with increased mRNAs of the ubiquitin-proteasome proteolytic pathway in human skeletal muscleG Tiao, S Hobler, J J Wang, et al.Respiratory Care Clinics of North America|September 1, 1996
Management of blunt chest injuryM Ferguson, F A LuchetteThe Journal of Trauma|May 25, 1999
Adrenergic antagonists reduce lactic acidosis in response to hemorrhagic shockF A Luchette, B R Robinson, L A Friend, et al.Surgery|August 1, 1981
Nitrogen-sparing mechanisms of singly administered branched-chain amino acids in the injured ratH R Freund, J H James, J E FischerAnnals of Surgery|April 1, 1986
Inhibited muscle amino acid uptake in sepsisP O Hasselgren, J H James, J E FischerPageof 52