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Proceedings of the National Academy of Sciences of the United States of America|September 14, 2000
Mutational inactivation of the proapoptotic gene BAX confers selective advantage during tumor clonal evolutionY Ionov, H Yamamoto, S Krajewski, et al.
The Journal of Biological Chemistry|May 22, 2001
X-linked inhibitor of apoptosis protein (XIAP) inhibits caspase-3 and -7 in distinct modesY Suzuki, Y Nakabayashi, K Nakata, et al.
Journal of Cellular Biochemistry|July 8, 2010
Co-chaperone BAG3 and adenovirus penton base protein partnershipE Gout, M Gutkowska, S Takayama, et al.
The Journal of Biological Chemistry|April 18, 1998
Granzyme release and caspase activation in activated human T-lymphocytesJ M Zapata, R Takahashi, G S Salvesen, et al.
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|August 13, 1998
Bcl-x(l) Bax interaction after transient global ischemiaF J Antonawich, S Krajewski, J C Reed, et al.
Biochemical and Biophysical Research Communications|April 28, 1997
Bcl-2 relieves the trans-repressive function of the glucocorticoid receptor and inhibits the activation of CPP32-like cysteine proteasesT Miyashita, U Mami, T Inoue, et al.
Molecular and Cellular Biology|February 12, 2000
The putative pore-forming domain of Bax regulates mitochondrial localization and interaction with Bcl-X(L)S Nouraini, E Six, S Matsuyama, et al.
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