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European Journal of Pharmacology|August 15, 1989
Facilitation of [3H]acetylcholine and [3H]5-hydroxytryptamine release from rat cerebral cortex synaptosomes by a factor extracted from the skin of the Australian frog Pseudophryne coriaceaM Raiteri, M Marchi, G Bonanno, et al.
European Journal of Pharmacology|March 17, 1992
Dopamine release and dopaminergic inhibition of acetylcholine release in rat striatal slices after nigro-striatal hemitransection and parenteral ganglioside administrationM Raiteri, M Marchi, G Bonanno, et al.
Journal of Neurochemistry|July 1, 1993
Glutamic acid and gamma-aminobutyric acid modulate each other's release through heterocarriers sited on the axon terminals of rat brainG Bonanno, A Pittaluga, E Fedele, et al.
European Journal of Pharmacology|June 24, 1993
CGP 52432: a novel potent and selective GABAB autoreceptor antagonist in rat cerebral cortexM Lanza, A Fassio, A Gemignani, et al.
Neurochemistry International|May 22, 2010
Carrier-mediated and carrier-independent release of serotonin from isolated central nerve endingsG Maura, A Gemignani, P Versace, et al.
British Journal of Pharmacology|February 12, 1998
Activation of brain nitric oxide synthase in depolarized human temporal cortex slices: differential role of voltage-sensitive calcium channelsG Fontana, E Fedele, M Cossu, et al.
The Journal of Pharmacology and Experimental Therapeutics|January 1, 1991
gamma-Aminobutyric acid (GABA) stimulates somatostatin release following activation of a GABA uptake carrier located on somatostatin nerve endings of rat cerebral cortexM Raiteri, G Bonanno, E Fedele, et al.
Neuropharmacology|December 10, 1999
Selective block of rat and human neocortex GABA(B) receptors regulating somatostatin release by a GABA(B) antagonist endowed with cognition enhancing activityG Bonanno, F Carità, P Cavazzani, et al.
Journal of Neurochemistry|January 5, 2000
Entrapping of impermeant probes of different size into nonpermeabilized synaptosomes as a method to study presynaptic mechanismsM Raiteri, R Sala, A Fassio, et al.
European Journal of Pharmacology|January 5, 1999
GABA(B) receptors as potential targets for drugs able to prevent excessive excitatory amino acid transmission in the spinal cordG Bonanno, A Fassio, R Sala, et al.
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