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F Checler

Showing results (91-100 of 120) with videos related to

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Neuroscience|November 24, 2015
Sox2 functionally interacts with βAPP, the βAPP intracellular domain and ADAM10 at a transcriptional level in human cellsG Sarlak, H H Htoo, J-F Hernandez, et al.
Molecular Psychiatry|September 21, 2016
The transcription factor XBP1s restores hippocampal synaptic plasticity and memory by control of the Kalirin-7 pathway in Alzheimer modelM Cissé, E Duplan, T Lorivel, et al.
The Journal of Biological Chemistry|July 31, 2001
The disintegrins ADAM10 and TACE contribute to the constitutive and phorbol ester-regulated normal cleavage of the cellular prion proteinB Vincent, E Paitel, P Saftig, et al.
European Journal of Biochemistry|October 19, 2001
The C-terminal fragment of the Alzheimer's disease amyloid protein precursor is degraded by a proteasome-dependent mechanism distinct from gamma-secretaseJ Nunan, M S Shearman, F Checler, et al.
Annales De Biologie Clinique|October 1, 1998
[Strategies for identification of secretases implicated in Alzheimer's disease]P Marambaud, N Chevallier, E Lopez-Perez, et al.
Journal of Alzheimer'S Disease : JAD|September 6, 2002
Apoptotic neurons in Alzheimer's disease frequently show intracellular Abeta42 labelingD.H. Chui, E. Dobo, T. Makifuchi, et al.
Biochimie|October 1, 1983
[Radioactive analogs of neurotensin. II. Preparation of tritiated neurotensin from a synthetic multi-unsaturated derivative]C Labbé-Jullié, D Blanot, J L Morgat, et al.
Journal of Medicinal Chemistry|May 14, 1993
Synthesis and analgesic effects of N-[3-[(hydroxyamino) carbonyl]-1-oxo-2(R)-benzylpropyl]-L-isoleucyl-L-leucine, a new potent inhibitor of multiple neurotensin/neuromedin N degrading enzymesS Doulut, I Dubuc, M Rodriguez, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 20, 1999
Endoplasmic reticulum and trans-Golgi network generate distinct populations of Alzheimer beta-amyloid peptidesJ P Greenfield, J Tsai, G K Gouras, et al.
Human Molecular Genetics|August 7, 2001
Pathogenic APP mutations near the gamma-secretase cleavage site differentially affect Abeta secretion and APP C-terminal fragment stabilityC De Jonghe, C Esselens, S Kumar-Singh, et al.
Pageof 12

Showing results (91-100 of 120) with videos related to

Sort By:
Pageof 12
Neuroscience|November 24, 2015
Sox2 functionally interacts with βAPP, the βAPP intracellular domain and ADAM10 at a transcriptional level in human cellsG Sarlak, H H Htoo, J-F Hernandez, et al.
Molecular Psychiatry|September 21, 2016
The transcription factor XBP1s restores hippocampal synaptic plasticity and memory by control of the Kalirin-7 pathway in Alzheimer modelM Cissé, E Duplan, T Lorivel, et al.
The Journal of Biological Chemistry|July 31, 2001
The disintegrins ADAM10 and TACE contribute to the constitutive and phorbol ester-regulated normal cleavage of the cellular prion proteinB Vincent, E Paitel, P Saftig, et al.
European Journal of Biochemistry|October 19, 2001
The C-terminal fragment of the Alzheimer's disease amyloid protein precursor is degraded by a proteasome-dependent mechanism distinct from gamma-secretaseJ Nunan, M S Shearman, F Checler, et al.
Annales De Biologie Clinique|October 1, 1998
[Strategies for identification of secretases implicated in Alzheimer's disease]P Marambaud, N Chevallier, E Lopez-Perez, et al.
Journal of Alzheimer'S Disease : JAD|September 6, 2002
Apoptotic neurons in Alzheimer's disease frequently show intracellular Abeta42 labelingD.H. Chui, E. Dobo, T. Makifuchi, et al.
Biochimie|October 1, 1983
[Radioactive analogs of neurotensin. II. Preparation of tritiated neurotensin from a synthetic multi-unsaturated derivative]C Labbé-Jullié, D Blanot, J L Morgat, et al.
Journal of Medicinal Chemistry|May 14, 1993
Synthesis and analgesic effects of N-[3-[(hydroxyamino) carbonyl]-1-oxo-2(R)-benzylpropyl]-L-isoleucyl-L-leucine, a new potent inhibitor of multiple neurotensin/neuromedin N degrading enzymesS Doulut, I Dubuc, M Rodriguez, et al.
Proceedings of the National Academy of Sciences of the United States of America|January 20, 1999
Endoplasmic reticulum and trans-Golgi network generate distinct populations of Alzheimer beta-amyloid peptidesJ P Greenfield, J Tsai, G K Gouras, et al.
Human Molecular Genetics|August 7, 2001
Pathogenic APP mutations near the gamma-secretase cleavage site differentially affect Abeta secretion and APP C-terminal fragment stabilityC De Jonghe, C Esselens, S Kumar-Singh, et al.
Pageof 12