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F E Jensen

Showing results (21-30 of 28) with videos related to

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Journal of Neurophysiology|February 21, 1998
Acute and chronic increases in excitability in rat hippocampal slices after perinatal hypoxia In vivoF E Jensen, C Wang, C E Stafstrom, et al.
Neuroscience|September 1, 1994
The putative essential nutrient pyrroloquinoline quinone is neuroprotective in a rodent model of hypoxic/ischemic brain injuryF E Jensen, G J Gardner, A P Williams, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 6, 2001
Decreased glutamate receptor 2 expression and enhanced epileptogenesis in immature rat hippocampus after perinatal hypoxia-induced seizuresR M Sanchez, S Koh, C Rio, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|March 8, 2000
Novel role for the NMDA receptor redox modulatory site in the pathophysiology of seizuresR M Sanchez, C Wang, G Gardner, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 1, 1992
Open-channel block of N-methyl-D-aspartate (NMDA) responses by memantine: therapeutic advantage against NMDA receptor-mediated neurotoxicityH S Chen, J W Pellegrini, S K Aggarwal, et al.
Neuropathology and Applied Neurobiology|January 29, 2008
Minocycline treatment following hypoxic/ischaemic injury attenuates white matter injury in a rodent model of periventricular leucomalaciaM Lechpammer, S M Manning, F Samonte, et al.
Neurology Research International|April 25, 2012
Regional differences in susceptibility to hypoxic-ischemic injury in the preterm brain: exploring the spectrum from white matter loss to selective grey matter injury in a rat modelD B Selip, L L Jantzie, M Chang, et al.
Neuroscience|August 11, 1998
Neuroprotective concentrations of the N-methyl-D-aspartate open-channel blocker memantine are effective without cytoplasmic vacuolation following post-ischemic administration and do not block maze learning or long-term potentiationH S Chen, Y F Wang, P V Rayudu, et al.
Pageof 3

Showing results (21-30 of 28) with videos related to

Sort By:
Pageof 3
You have reached the last page of results.This site can display upto 28 results.
Journal of Neurophysiology|February 21, 1998
Acute and chronic increases in excitability in rat hippocampal slices after perinatal hypoxia In vivoF E Jensen, C Wang, C E Stafstrom, et al.
Neuroscience|September 1, 1994
The putative essential nutrient pyrroloquinoline quinone is neuroprotective in a rodent model of hypoxic/ischemic brain injuryF E Jensen, G J Gardner, A P Williams, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 6, 2001
Decreased glutamate receptor 2 expression and enhanced epileptogenesis in immature rat hippocampus after perinatal hypoxia-induced seizuresR M Sanchez, S Koh, C Rio, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|March 8, 2000
Novel role for the NMDA receptor redox modulatory site in the pathophysiology of seizuresR M Sanchez, C Wang, G Gardner, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 1, 1992
Open-channel block of N-methyl-D-aspartate (NMDA) responses by memantine: therapeutic advantage against NMDA receptor-mediated neurotoxicityH S Chen, J W Pellegrini, S K Aggarwal, et al.
Neuropathology and Applied Neurobiology|January 29, 2008
Minocycline treatment following hypoxic/ischaemic injury attenuates white matter injury in a rodent model of periventricular leucomalaciaM Lechpammer, S M Manning, F Samonte, et al.
Neurology Research International|April 25, 2012
Regional differences in susceptibility to hypoxic-ischemic injury in the preterm brain: exploring the spectrum from white matter loss to selective grey matter injury in a rat modelD B Selip, L L Jantzie, M Chang, et al.
Neuroscience|August 11, 1998
Neuroprotective concentrations of the N-methyl-D-aspartate open-channel blocker memantine are effective without cytoplasmic vacuolation following post-ischemic administration and do not block maze learning or long-term potentiationH S Chen, Y F Wang, P V Rayudu, et al.
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