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Blood|March 31, 2005
Quiescence and functional reprogramming of Epstein-Barr virus (EBV)-specific CD8+ T cells during persistent infectionPádraic J Dunne, Lavina Belaramani, Jean M Fletcher, et al.
Cancer Research|July 20, 2017
Deubiquitinating Enzyme USP9X Suppresses Tumor Growth via LATS Kinase and Core Components of the Hippo PathwayAleksandra Toloczko, Fusheng Guo, Hiu-Fung Yuen, et al.
Frontiers in Endocrinology|February 24, 2016
Feeding Problems Are Persistent in Children with Severe Congenital HyperinsulinismIndraneel Banerjee, Lynette Forsythe, Mars Skae, et al.
Endocrine-Related Cancer|January 25, 2003
Uncontrolled insulin secretion from a childhood pancreatic beta-cell adenoma is not due to the functional loss of ATP-sensitive potassium channelsK Hussain, K E Cosgrove, R M Shepherd, et al.
BMC Public Health|March 16, 2020
Assertive outreach treatment versus care as usual for the treatment of high-need, high-cost alcohol related frequent attenders: study protocol for a randomised controlled trialR Blackwood, A Wolstenholme, A Kimergård, et al.
Archives of Disease in Childhood|May 1, 1996
Ionic control of beta cell function in nesidioblastosis. A possible therapeutic role for calcium channel blockadeK J Lindley, M J Dunne, C Kane, et al.
The Journal of Clinical Endocrinology and Metabolism|June 13, 2017
Atypical Forms of Congenital Hyperinsulinism in Infancy Are Associated With Mosaic Patterns of Immature Islet CellsBing Han, Zainab Mohamed, Maria Salomon Estebanez, et al.
Journal of Controlled Release : Official Journal of the Controlled Release Society|November 23, 2020
Rational design and characterisation of a linear cell penetrating peptide for non-viral gene deliveryEmma M McErlean, Monika Ziminska, Cian M McCrudden, et al.
Journal of Pediatric Endocrinology & Metabolism : JPEM|January 14, 2005
Infantile hyperinsulinism associated with enteropathy, deafness and renal tubulopathy: clinical manifestations of a syndrome caused by a contiguous gene deletion located on chromosome 11pKhalid Hussain, Maria Bitner-Glindzicz, Diana Blaydon, et al.
The Journal of Pediatrics|December 3, 2014
Increased plasma incretin concentrations identifies a subset of patients with persistent congenital hyperinsulinism without KATP channel gene defectsYanqin Shi, Hima B Avatapalle, Mars S Skae, et al.
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