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F M Ashcroft

Showing results (141-150 of 151) with videos related to

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The Journal of Biological Chemistry|April 10, 1999
Miniglucagon (glucagon 19-29), a potent and efficient inhibitor of secretagogue-induced insulin release through a Ca2+ pathwayS Dalle, P Smith, P Blache, et al.
FEBS Letters|June 26, 1995
Characterization and variation of a human inwardly-rectifying-K-channel gene (KCNJ6): a putative ATP-sensitive K-channel subunitH Sakura, C Bond, M Warren-Perry, et al.
Physiology & Behavior|March 15, 2006
Behavioral phenotyping of mice lacking the K ATP channel subunit Kir6.2R M J Deacon, R C Brook, D Meyer, et al.
The EMBO Journal|June 17, 1998
Molecular determinants of KATP channel inhibition by ATPS J Tucker, F M Gribble, P Proks, et al.
Diabetes|May 1, 1995
No evidence for mutations in a putative beta-cell ATP-sensitive K+ channel subunit in MODY, NIDDM, or GDMY Zhang, M Warren-Perry, H Sakura, et al.
British Journal of Pharmacology|November 11, 1999
In vitro mechanism of action on insulin release of S-22068, a new putative antidiabetic compoundL Le Brigand, A Virsolvy, D Manechez, et al.
Nature|February 8, 1996
Promiscuous coupling between the sulphonylurea receptor and inwardly rectifying potassium channelsC Ammälä, A Moorhouse, F Gribble, et al.
FEBS Letters|April 9, 1990
Expression of voltage-gated K+ channels in insulin-producing cells. Analysis by polymerase chain reactionC Betsholtz, A Baumann, S Kenna, et al.
Diabetologia|March 13, 2008
A Kir6.2 mutation causing severe functional effects in vitro produces neonatal diabetes without the expected neurological complicationsP Tammaro, S E Flanagan, B Zadek, et al.
Diabetes|May 20, 1999
A point mutation inactivating the sulfonylurea receptor causes the severe form of persistent hyperinsulinemic hypoglycemia of infancy in FinlandT Otonkoski, C Ammälä, H Huopio, et al.
Pageof 16

Showing results (141-150 of 151) with videos related to

Sort By:
Pageof 16
The Journal of Biological Chemistry|April 10, 1999
Miniglucagon (glucagon 19-29), a potent and efficient inhibitor of secretagogue-induced insulin release through a Ca2+ pathwayS Dalle, P Smith, P Blache, et al.
FEBS Letters|June 26, 1995
Characterization and variation of a human inwardly-rectifying-K-channel gene (KCNJ6): a putative ATP-sensitive K-channel subunitH Sakura, C Bond, M Warren-Perry, et al.
Physiology & Behavior|March 15, 2006
Behavioral phenotyping of mice lacking the K ATP channel subunit Kir6.2R M J Deacon, R C Brook, D Meyer, et al.
The EMBO Journal|June 17, 1998
Molecular determinants of KATP channel inhibition by ATPS J Tucker, F M Gribble, P Proks, et al.
Diabetes|May 1, 1995
No evidence for mutations in a putative beta-cell ATP-sensitive K+ channel subunit in MODY, NIDDM, or GDMY Zhang, M Warren-Perry, H Sakura, et al.
British Journal of Pharmacology|November 11, 1999
In vitro mechanism of action on insulin release of S-22068, a new putative antidiabetic compoundL Le Brigand, A Virsolvy, D Manechez, et al.
Nature|February 8, 1996
Promiscuous coupling between the sulphonylurea receptor and inwardly rectifying potassium channelsC Ammälä, A Moorhouse, F Gribble, et al.
FEBS Letters|April 9, 1990
Expression of voltage-gated K+ channels in insulin-producing cells. Analysis by polymerase chain reactionC Betsholtz, A Baumann, S Kenna, et al.
Diabetologia|March 13, 2008
A Kir6.2 mutation causing severe functional effects in vitro produces neonatal diabetes without the expected neurological complicationsP Tammaro, S E Flanagan, B Zadek, et al.
Diabetes|May 20, 1999
A point mutation inactivating the sulfonylurea receptor causes the severe form of persistent hyperinsulinemic hypoglycemia of infancy in FinlandT Otonkoski, C Ammälä, H Huopio, et al.
Pageof 16