Showing results (11-20 of 38) with videos related to

Sort By:
Pageof 4
The Journal of Biological Chemistry|October 15, 1988
Purification and properties of a single-chain urokinase-type plasminogen activator form produced by subcultured human umbilical vein endothelial cellsF M Booyse, P H Lin, M Traylor, et al.
Thrombosis and Haemostasis|February 29, 1976
Comparison of two types of endothelial cells in long term cultureB J Sedlak, F M Booyse, S Bell, et al.
The Journal of Biological Chemistry|October 15, 1988
Isolation and interrelationships of the multiple molecular tissue-type and urokinase-type plasminogen activator forms produced by cultured human umbilical vein endothelial cellsF M Booyse, J Scheinbuks, P H Lin, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|June 10, 2000
Hypertriglyceridemic VLDL downregulates tissue plasminogen activator gene transcription through cis-repressive region(s) in the tissue plasminogen activator promoter in cultured human endothelial cellsE M Tabengwa, R L Benza, H E Grenett, et al.
Seminars in Thrombosis and Hemostasis|July 1, 1986
Rapid release and deactivation of plasminogen activators in human endothelial cell cultures in the presence of thrombin and ionophore A23187F M Booyse, R Bruce, D Dolenak, et al.
Biochimica Et Biophysica Acta|January 23, 1976
Adenosine cyclic 3',5'-monophosphate-dependent protein kinase from human plateletsF M Booyse, J Marr, D C Yang, et al.
Thrombosis Research|May 1, 1990
Lipolyzed hypertriglyceridemic serum and triglyceride-rich lipoprotein cause lipid accumulation in and are cytotoxic to cultured human endothelial cells. High density lipoproteins inhibit this cytotoxicityM T Speidel, F M Booyse, A Abrams, et al.
Thrombosis Research|February 16, 2000
Ethanol downregulates transcription of the PAI-1 gene in cultured human endothelial cellsH E Grenett, M L Aikens, E M Tabengwa, et al.
The American Journal of Physiology|June 1, 1989
Hypoxia stimulates endothelial cell angiotensin-converting enzyme antigen synthesisS J King, F M Booyse, P H Lin, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 1, 1987
Expression of abnormal von Willebrand factor by endothelial cells from a patient with type IIA von Willebrand diseaseR B Levene, F M Booyse, J Chediak, et al.
Pageof 4