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F RAPPAPORT

Showing results (61-70 of 84) with videos related to

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Oncogene|July 4, 2002
MLL-SEPTIN6 fusion recurs in novel translocation of chromosomes 3, X, and 11 in infant acute myelomonocytic leukaemia and in t(X;11) in infant acute myeloid leukaemia, and MLL genomic breakpoint in complex MLL-SEPTIN6 rearrangement is a DNA topoisomerase II cleavage siteDiana J Slater, Eva Hilgenfeld, Eric F Rappaport, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 30, 1998
t(11;22)(q23;q11.2) In acute myeloid leukemia of infant twins fuses MLL with hCDCrel, a cell division cycle gene in the genomic region of deletion in DiGeorge and velocardiofacial syndromesM D Megonigal, E F Rappaport, D H Jones, et al.
Biochemistry|February 15, 2001
Etoposide metabolites enhance DNA topoisomerase II cleavage near leukemia-associated MLL translocation breakpointsB D Lovett, D Strumberg, I A Blair, et al.
Pediatric Blood & Cancer|March 22, 2016
Unique Familial MLL(KMT2A)-Rearranged Precursor B-Cell Infant Acute Lymphoblastic Leukemia in Non-twin SiblingsKaren A Urtishak, Blaine W Robinson, Eric F Rappaport, et al.
Blood|January 7, 1998
Panhandle polymerase chain reaction amplifies MLL genomic translocation breakpoint involving unknown partner geneC A Felix, C S Kim, M D Megonigal, et al.
Genome Research|April 8, 2017
Genome-wide TOP2A DNA cleavage is biased toward translocated and highly transcribed lociXiang Yu, James W Davenport, Karen A Urtishak, et al.
Genes, Brain, and Behavior|December 7, 2010
Quantitative trait loci for electrical seizure threshold mapped in C57BLKS/J and C57BL/10SnJ miceT N Ferraro, G G Smith, D Ballard, et al.
Diabetologia|August 20, 2016
The type 2 diabetes presumed causal variant within TCF7L2 resides in an element that controls the expression of ACSL5Qianghua Xia, Alessandra Chesi, Elisabetta Manduchi, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 8, 2000
Detection of leukemia-associated MLL-GAS7 translocation early during chemotherapy with DNA topoisomerase II inhibitorsM D Megonigal, N K Cheung, E F Rappaport, et al.
Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology|September 5, 2008
Proline affects brain function in 22q11DS children with the low activity COMT 158 alleleJacob A S Vorstman, Bruce I Turetsky, Monique E J Sijmens-Morcus, et al.
Pageof 9

Showing results (61-70 of 84) with videos related to

Sort By:
Pageof 9
Oncogene|July 4, 2002
MLL-SEPTIN6 fusion recurs in novel translocation of chromosomes 3, X, and 11 in infant acute myelomonocytic leukaemia and in t(X;11) in infant acute myeloid leukaemia, and MLL genomic breakpoint in complex MLL-SEPTIN6 rearrangement is a DNA topoisomerase II cleavage siteDiana J Slater, Eva Hilgenfeld, Eric F Rappaport, et al.
Proceedings of the National Academy of Sciences of the United States of America|May 30, 1998
t(11;22)(q23;q11.2) In acute myeloid leukemia of infant twins fuses MLL with hCDCrel, a cell division cycle gene in the genomic region of deletion in DiGeorge and velocardiofacial syndromesM D Megonigal, E F Rappaport, D H Jones, et al.
Biochemistry|February 15, 2001
Etoposide metabolites enhance DNA topoisomerase II cleavage near leukemia-associated MLL translocation breakpointsB D Lovett, D Strumberg, I A Blair, et al.
Pediatric Blood & Cancer|March 22, 2016
Unique Familial MLL(KMT2A)-Rearranged Precursor B-Cell Infant Acute Lymphoblastic Leukemia in Non-twin SiblingsKaren A Urtishak, Blaine W Robinson, Eric F Rappaport, et al.
Blood|January 7, 1998
Panhandle polymerase chain reaction amplifies MLL genomic translocation breakpoint involving unknown partner geneC A Felix, C S Kim, M D Megonigal, et al.
Genome Research|April 8, 2017
Genome-wide TOP2A DNA cleavage is biased toward translocated and highly transcribed lociXiang Yu, James W Davenport, Karen A Urtishak, et al.
Genes, Brain, and Behavior|December 7, 2010
Quantitative trait loci for electrical seizure threshold mapped in C57BLKS/J and C57BL/10SnJ miceT N Ferraro, G G Smith, D Ballard, et al.
Diabetologia|August 20, 2016
The type 2 diabetes presumed causal variant within TCF7L2 resides in an element that controls the expression of ACSL5Qianghua Xia, Alessandra Chesi, Elisabetta Manduchi, et al.
Proceedings of the National Academy of Sciences of the United States of America|March 8, 2000
Detection of leukemia-associated MLL-GAS7 translocation early during chemotherapy with DNA topoisomerase II inhibitorsM D Megonigal, N K Cheung, E F Rappaport, et al.
Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology|September 5, 2008
Proline affects brain function in 22q11DS children with the low activity COMT 158 alleleJacob A S Vorstman, Bruce I Turetsky, Monique E J Sijmens-Morcus, et al.
Pageof 9