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F Saudou

Showing results (11-20 of 20) with videos related to

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Behavioural Brain Research|January 1, 1996
5-HT1B receptor knock out--behavioral consequencesS Ramboz, F Saudou, D A Amara, et al.
Nature|November 23, 1995
Polyglutamine expansion as a pathological epitope in Huntington's disease and four dominant cerebellar ataxiasY Trottier, Y Lutz, G Stevanin, et al.
Molecular and Cellular Biology|August 26, 1998
Fos family members induce cell cycle entry by activating cyclin D1J R Brown, E Nigh, R J Lee, et al.
Nature Genetics|May 1, 1995
Cellular localization of the Huntington's disease protein and discrimination of the normal and mutated formY Trottier, D Devys, G Imbert, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 26, 1998
Essential role of the fosB gene in molecular, cellular, and behavioral actions of chronic electroconvulsive seizuresN Hiroi, G J Marek, J R Brown, et al.
Annals of Neurology|December 10, 1997
Differential distribution of the normal and mutated forms of huntingtin in the human brainI Gourfinkel-An, G Cancel, Y Trottier, et al.
Nature Genetics|November 1, 1996
Cloning of the gene for spinocerebellar ataxia 2 reveals a locus with high sensitivity to expanded CAG/glutamine repeatsG Imbert, F Saudou, G Yvert, et al.
Nature Genetics|September 1, 1997
Cloning of the SCA7 gene reveals a highly unstable CAG repeat expansionG David, N Abbas, G Stevanin, et al.
Human Molecular Genetics|December 1, 1996
Screening for proteins with polyglutamine expansions in autosomal dominant cerebellar ataxiasG Stevanin, Y Trottier, G Cancel, et al.
Human Molecular Genetics|May 1, 1997
Molecular and clinical correlations in spinocerebellar ataxia 2: a study of 32 familiesG Cancel, A Dürr, O Didierjean, et al.
Pageof 2

Showing results (11-20 of 20) with videos related to

Sort By:
Pageof 2
You have reached the last page of results.This site can display upto 20 results.
Behavioural Brain Research|January 1, 1996
5-HT1B receptor knock out--behavioral consequencesS Ramboz, F Saudou, D A Amara, et al.
Nature|November 23, 1995
Polyglutamine expansion as a pathological epitope in Huntington's disease and four dominant cerebellar ataxiasY Trottier, Y Lutz, G Stevanin, et al.
Molecular and Cellular Biology|August 26, 1998
Fos family members induce cell cycle entry by activating cyclin D1J R Brown, E Nigh, R J Lee, et al.
Nature Genetics|May 1, 1995
Cellular localization of the Huntington's disease protein and discrimination of the normal and mutated formY Trottier, D Devys, G Imbert, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 26, 1998
Essential role of the fosB gene in molecular, cellular, and behavioral actions of chronic electroconvulsive seizuresN Hiroi, G J Marek, J R Brown, et al.
Annals of Neurology|December 10, 1997
Differential distribution of the normal and mutated forms of huntingtin in the human brainI Gourfinkel-An, G Cancel, Y Trottier, et al.
Nature Genetics|November 1, 1996
Cloning of the gene for spinocerebellar ataxia 2 reveals a locus with high sensitivity to expanded CAG/glutamine repeatsG Imbert, F Saudou, G Yvert, et al.
Nature Genetics|September 1, 1997
Cloning of the SCA7 gene reveals a highly unstable CAG repeat expansionG David, N Abbas, G Stevanin, et al.
Human Molecular Genetics|December 1, 1996
Screening for proteins with polyglutamine expansions in autosomal dominant cerebellar ataxiasG Stevanin, Y Trottier, G Cancel, et al.
Human Molecular Genetics|May 1, 1997
Molecular and clinical correlations in spinocerebellar ataxia 2: a study of 32 familiesG Cancel, A Dürr, O Didierjean, et al.
Pageof 2