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F Starmer

Showing results (81-90 of 108) with videos related to

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Journal of Molecular and Cellular Cardiology|February 1, 1991
Lidocaine blockade of continuously and transiently accessible sites in cardiac sodium channelsC F Starmer, V V Nesterenko, A I Undrovinas, et al.
The American Journal of Physiology|October 1, 1990
Extracellular pH modulates block of both sodium and calcium channels by nicardipineF R Gilliam, P A Rivas, D J Wendt, et al.
Circulation|March 1, 1980
Clinical judgment and statistics. Lessons from a simulated randomized trial in coronary artery diseaseK L Lee, J F McNeer, C F Starmer, et al.
Circulation|April 1, 1973
Blood-gas and hemodynamic responses to oxygen in acute myocardial infarctionR M Davidson, B W Ramo, A G Wallace, et al.
Molecular Pharmacology|December 1, 1996
The role of inactivation in open-channel block of the sodium channel: studies with inactivation-deficient mutant channelsA O Grant, J E John, V V Nesterenko, et al.
The Journal of Pharmacology and Experimental Therapeutics|July 1, 1986
pKa does not predict pH potentiation of sodium channel blockade by lidocaine and W6211 in guinea pig ventricular myocardiumJ R Moorman, R Yee, T Bjornsson, et al.
The American Journal of Physiology|November 1, 1989
Ethacizin blockade of calcium channels: a test of the guarded receptor hypothesisC F Starmer, A I Undrovinas, F Scamps, et al.
Circulation|October 1, 1970
Hemodynamic findings in 123 patients with acute myocardial infarction on admissionB W Ramo, N Myers, A G Wallace, et al.
Circulation Research|October 1, 1984
Lipid solubility modulates pH potentiation of local anesthetic block of Vmax reactivation in guinea pig myocardiumA Broughton, A O Grant, C F Starmer, et al.
The American Journal of Physiology|December 1, 1992
Cellular Vmax reflects both membrane properties and the load presented by adjoining cellsM S Spach, J F Heidlage, E R Darken, et al.
Pageof 11

Showing results (81-90 of 108) with videos related to

Sort By:
Pageof 11
Journal of Molecular and Cellular Cardiology|February 1, 1991
Lidocaine blockade of continuously and transiently accessible sites in cardiac sodium channelsC F Starmer, V V Nesterenko, A I Undrovinas, et al.
The American Journal of Physiology|October 1, 1990
Extracellular pH modulates block of both sodium and calcium channels by nicardipineF R Gilliam, P A Rivas, D J Wendt, et al.
Circulation|March 1, 1980
Clinical judgment and statistics. Lessons from a simulated randomized trial in coronary artery diseaseK L Lee, J F McNeer, C F Starmer, et al.
Circulation|April 1, 1973
Blood-gas and hemodynamic responses to oxygen in acute myocardial infarctionR M Davidson, B W Ramo, A G Wallace, et al.
Molecular Pharmacology|December 1, 1996
The role of inactivation in open-channel block of the sodium channel: studies with inactivation-deficient mutant channelsA O Grant, J E John, V V Nesterenko, et al.
The Journal of Pharmacology and Experimental Therapeutics|July 1, 1986
pKa does not predict pH potentiation of sodium channel blockade by lidocaine and W6211 in guinea pig ventricular myocardiumJ R Moorman, R Yee, T Bjornsson, et al.
The American Journal of Physiology|November 1, 1989
Ethacizin blockade of calcium channels: a test of the guarded receptor hypothesisC F Starmer, A I Undrovinas, F Scamps, et al.
Circulation|October 1, 1970
Hemodynamic findings in 123 patients with acute myocardial infarction on admissionB W Ramo, N Myers, A G Wallace, et al.
Circulation Research|October 1, 1984
Lipid solubility modulates pH potentiation of local anesthetic block of Vmax reactivation in guinea pig myocardiumA Broughton, A O Grant, C F Starmer, et al.
The American Journal of Physiology|December 1, 1992
Cellular Vmax reflects both membrane properties and the load presented by adjoining cellsM S Spach, J F Heidlage, E R Darken, et al.
Pageof 11