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F Tagliavini

Showing results (91-100 of 133) with videos related to

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Proceedings of the National Academy of Sciences of the United States of America|October 15, 1993
Synthetic peptides homologous to prion protein residues 106-147 form amyloid-like fibrils in vitroF Tagliavini, F Prelli, L Verga, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 1, 1992
Prion protein preamyloid and amyloid deposits in Gerstmann-Sträussler-Scheinker disease, Indiana kindredG Giaccone, L Verga, O Bugiani, et al.
Biochemical and Biophysical Research Communications|August 16, 1993
Molecular characteristics of a protease-resistant, amyloidogenic and neurotoxic peptide homologous to residues 106-126 of the prion proteinC Selvaggini, L De Gioia, L Cantù, et al.
Neuroreport|April 20, 1999
Microglial cells respond to amyloidogenic PrP peptide by the production of inflammatory cytokinesJ M Peyrin, C I Lasmézas, S Haïk, et al.
Brain Pathology (Zurich, Switzerland)|April 1, 1996
Prion protein amyloidosisB Ghetti, P Piccardo, B Frangione, et al.
Brain Research|July 9, 1993
A68 is a component of paired helical filaments of Gerstmann-Sträussler-Scheinker disease, Indiana kindredF Tagliavini, G Giaccone, F Prelli, et al.
Clinical Neurophysiology : Official Journal of the International Federation of Clinical Neurophysiology|March 29, 2005
FVEPs in Creutzfeldt-Jacob disease: waveforms and interaction with the periodic EEG pattern assessed by single sweep analysisE Visani, P Agazzi, V Scaioli, et al.
Journal of Neuropathology and Experimental Neurology|November 1, 1996
Proteinase-K-resistant prion protein isoforms in Gerstmann-Sträussler-Scheinker disease (Indiana kindred)P Piccardo, C Seiler, S R Dlouhy, et al.
Cell|November 18, 1994
Amyloid fibrils in Gerstmann-Sträussler-Scheinker disease (Indiana and Swedish kindreds) express only PrP peptides encoded by the mutant alleleF Tagliavini, F Prelli, M Porro, et al.
Nature Medicine|April 1, 1995
Tau protein directly interacts with the amyloid beta-protein precursor: implications for Alzheimer's diseaseM A Smith, S L Siedlak, P L Richey, et al.
Pageof 14

Showing results (91-100 of 133) with videos related to

Sort By:
Pageof 14
Proceedings of the National Academy of Sciences of the United States of America|October 15, 1993
Synthetic peptides homologous to prion protein residues 106-147 form amyloid-like fibrils in vitroF Tagliavini, F Prelli, L Verga, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 1, 1992
Prion protein preamyloid and amyloid deposits in Gerstmann-Sträussler-Scheinker disease, Indiana kindredG Giaccone, L Verga, O Bugiani, et al.
Biochemical and Biophysical Research Communications|August 16, 1993
Molecular characteristics of a protease-resistant, amyloidogenic and neurotoxic peptide homologous to residues 106-126 of the prion proteinC Selvaggini, L De Gioia, L Cantù, et al.
Neuroreport|April 20, 1999
Microglial cells respond to amyloidogenic PrP peptide by the production of inflammatory cytokinesJ M Peyrin, C I Lasmézas, S Haïk, et al.
Brain Pathology (Zurich, Switzerland)|April 1, 1996
Prion protein amyloidosisB Ghetti, P Piccardo, B Frangione, et al.
Brain Research|July 9, 1993
A68 is a component of paired helical filaments of Gerstmann-Sträussler-Scheinker disease, Indiana kindredF Tagliavini, G Giaccone, F Prelli, et al.
Clinical Neurophysiology : Official Journal of the International Federation of Clinical Neurophysiology|March 29, 2005
FVEPs in Creutzfeldt-Jacob disease: waveforms and interaction with the periodic EEG pattern assessed by single sweep analysisE Visani, P Agazzi, V Scaioli, et al.
Journal of Neuropathology and Experimental Neurology|November 1, 1996
Proteinase-K-resistant prion protein isoforms in Gerstmann-Sträussler-Scheinker disease (Indiana kindred)P Piccardo, C Seiler, S R Dlouhy, et al.
Cell|November 18, 1994
Amyloid fibrils in Gerstmann-Sträussler-Scheinker disease (Indiana and Swedish kindreds) express only PrP peptides encoded by the mutant alleleF Tagliavini, F Prelli, M Porro, et al.
Nature Medicine|April 1, 1995
Tau protein directly interacts with the amyloid beta-protein precursor: implications for Alzheimer's diseaseM A Smith, S L Siedlak, P L Richey, et al.
Pageof 14