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Showing results (71-80 of 133) with videos related to

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The Journal of Biological Chemistry|May 24, 2000
Substitutions at codon 22 of Alzheimer's abeta peptide induce diverse conformational changes and apoptotic effects in human cerebral endothelial cellsL Miravalle, T Tokuda, R Chiarle, et al.
Sleep|December 1, 1995
Precocious loss of physiological sleep in a case of Creutzfeldt Jakob disease: a serial polygraphic studyM G Terzano, L Parrino, V Pietrini, et al.
Brain Pathology (Zurich, Switzerland)|January 1, 1995
Gerstmann-Sträussler-Scheinker disease and the Indiana kindredB Ghetti, S R Dlouhy, G Giaccone, et al.
Journal of Neuropathology and Experimental Neurology|January 1, 1993
Beta PP participates in PrP-amyloid plaques of Gerstmann-Sträussler-Scheinker disease, Indiana kindredO Bugiani, G Giaccone, L Verga, et al.
The Journal of Biological Chemistry|March 18, 1994
Conformational polymorphism of the amyloidogenic and neurotoxic peptide homologous to residues 106-126 of the prion proteinL De Gioia, C Selvaggini, E Ghibaudi, et al.
The Journal of Biological Chemistry|April 24, 2001
The stimulation of inducible nitric-oxide synthase by the prion protein fragment 106--126 in human microglia is tumor necrosis factor-alpha-dependent and involves p38 mitogen-activated protein kinaseC Fabrizi, V Silei, M Menegazzi, et al.
Molecular Neurobiology|January 20, 2018
In Situ Tissue Labeling of Cerebral Amyloid Using HIV-Related Tat PeptideE Maderna, L Colombo, A Cagnotto, et al.
Neuroscience Letters|August 5, 1991
Relationship between non-fibrillary amyloid precursors and cell processes in the cortical neuropil of Alzheimer patientsK H el Hachimi, L Verga, G Giaccone, et al.
Journal of Neuroscience Research|November 18, 1998
Prion protein fragment 106-126 induces apoptotic cell death and impairment of L-type voltage-sensitive calcium channel activity in the GH3 cell lineT Florio, S Thellung, C Amico, et al.
Neuropathology and Applied Neurobiology|September 3, 2011
Atypical tauopathy in a patient with LRRK2-G2019S mutation and tremor-dominant ParkinsonismC Ruffmann, G Giaccone, M Canesi, et al.
Pageof 14

Showing results (71-80 of 133) with videos related to

Sort By:
Pageof 14
The Journal of Biological Chemistry|May 24, 2000
Substitutions at codon 22 of Alzheimer's abeta peptide induce diverse conformational changes and apoptotic effects in human cerebral endothelial cellsL Miravalle, T Tokuda, R Chiarle, et al.
Sleep|December 1, 1995
Precocious loss of physiological sleep in a case of Creutzfeldt Jakob disease: a serial polygraphic studyM G Terzano, L Parrino, V Pietrini, et al.
Brain Pathology (Zurich, Switzerland)|January 1, 1995
Gerstmann-Sträussler-Scheinker disease and the Indiana kindredB Ghetti, S R Dlouhy, G Giaccone, et al.
Journal of Neuropathology and Experimental Neurology|January 1, 1993
Beta PP participates in PrP-amyloid plaques of Gerstmann-Sträussler-Scheinker disease, Indiana kindredO Bugiani, G Giaccone, L Verga, et al.
The Journal of Biological Chemistry|March 18, 1994
Conformational polymorphism of the amyloidogenic and neurotoxic peptide homologous to residues 106-126 of the prion proteinL De Gioia, C Selvaggini, E Ghibaudi, et al.
The Journal of Biological Chemistry|April 24, 2001
The stimulation of inducible nitric-oxide synthase by the prion protein fragment 106--126 in human microglia is tumor necrosis factor-alpha-dependent and involves p38 mitogen-activated protein kinaseC Fabrizi, V Silei, M Menegazzi, et al.
Molecular Neurobiology|January 20, 2018
In Situ Tissue Labeling of Cerebral Amyloid Using HIV-Related Tat PeptideE Maderna, L Colombo, A Cagnotto, et al.
Neuroscience Letters|August 5, 1991
Relationship between non-fibrillary amyloid precursors and cell processes in the cortical neuropil of Alzheimer patientsK H el Hachimi, L Verga, G Giaccone, et al.
Journal of Neuroscience Research|November 18, 1998
Prion protein fragment 106-126 induces apoptotic cell death and impairment of L-type voltage-sensitive calcium channel activity in the GH3 cell lineT Florio, S Thellung, C Amico, et al.
Neuropathology and Applied Neurobiology|September 3, 2011
Atypical tauopathy in a patient with LRRK2-G2019S mutation and tremor-dominant ParkinsonismC Ruffmann, G Giaccone, M Canesi, et al.
Pageof 14