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Cells|September 27, 2024
Structural and Functional Characterization of the Most Frequent Pathogenic PRKN Substitution p.R275WBernardo A Bustillos, Liam T Cocker, Mathew A Coban, et al.Autophagy Reports|February 26, 2025
Alterations of PINK1-PRKN signaling in mice during normal agingZahra Baninameh, Jens O Watzlawik, Xu Hou, et al.Biorxiv : the Preprint Server for Biology|May 15, 2024
Alterations of PINK1-PRKN signaling in mice during normal agingZahra Baninameh, Jens O Watzlawik, Xu Hou, et al.Medrxiv : the Preprint Server for Health Sciences|October 31, 2023
Genome-wide association study identifies <i>APOE</i> and <i>ZMIZ1</i> variants as mitophagy modifiers in Lewy body diseaseXu Hou, Michael G Heckman, Fabienne C Fiesel, et al.Proceedings of the National Academy of Sciences of the United States of America|January 28, 2025
Endogenous LRRK2 and PINK1 function in a convergent neuroprotective ciliogenesis pathway in the brainEnrico Bagnoli, Yu-En Lin, Sophie Burel, et al.Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association|May 1, 2025
Genome-wide association analysis identifies APOE as a mitophagy modifier in Lewy body diseaseXu Hou, Michael G Heckman, Fabienne C Fiesel, et al.Biorxiv : the Preprint Server for Biology|May 4, 2026
Phosphorylated ubiquitin is a secondary messenger and an epigenetic mark mediating mitochondria to nucleus signalingThomas J Mercer, Bence Daniel, Craig Fredrickson, et al.Cell Reports|January 26, 2017
PINK1 Primes Parkin-Mediated Ubiquitination of PARIS in Dopaminergic Neuronal SurvivalYunjong Lee, Daniel A Stevens, Sung-Ung Kang, et al.EMBO Reports|July 12, 2015
(Patho-)physiological relevance of PINK1-dependent ubiquitin phosphorylationFabienne C Fiesel, Maya Ando, Roman Hudec, et al.Alzheimer'S & Dementia : the Journal of the Alzheimer'S Association|October 22, 2020
Mitophagy alterations in Alzheimer's disease are associated with granulovacuolar degeneration and early tau pathologyXu Hou, Jens O Watzlawik, Casey Cook, et al.Pageof 7