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British Journal of Pharmacology|March 19, 2016
New aspects of p66Shc in ischaemia reperfusion injury and other cardiovascular diseasesFabio Di Lisa, Marco Giorgio, Peter Ferdinandy, et al.Journal of Molecular and Cellular Cardiology|September 22, 2007
Mitochondrial respiration and membrane potential after low-flow ischemia are not affected by ischemic preconditioningKerstin Boengler, Petra Gres, Giuliano Dodoni, et al.Journal of Cellular and Molecular Medicine|May 9, 2020
The role of mitochondrial reactive oxygen species, NO and H<sub>2</sub> S in ischaemia/reperfusion injury and cardioprotectionIoanna Andreadou, Rainer Schulz, Andreas Papapetropoulos, et al.Archives of Biochemistry and Biophysics|November 7, 2020
Effects of air pollution particles (ultrafine and fine particulate matter) on mitochondrial function and oxidative stress - Implications for cardiovascular and neurodegenerative diseasesAndreas Daiber, Marin Kuntic, Omar Hahad, et al.Journal of Cellular and Molecular Medicine|June 4, 2020
Mitochondrial ion channels as targets for cardioprotectionDerek J Hausenloy, Rainer Schulz, Henrique Girao, et al.Circulation Research|August 16, 2005
Impairment of diazoxide-induced formation of reactive oxygen species and loss of cardioprotection in connexin 43 deficient miceFrank R Heinzel, Yukun Luo, Xiaokui Li, et al.European Heart Journal|January 26, 2006
Oxidative modification of tropomyosin and myocardial dysfunction following coronary microembolizationMarcella Canton, Andreas Skyschally, Roberta Menabò, et al.Cardiovascular Research|June 20, 2006
Formation of reactive oxygen species at increased contraction frequency in rat cardiomyocytesFrank R Heinzel, Yukun Luo, Giuliano Dodoni, et al.Journal of Molecular and Cellular Cardiology|March 24, 2009
A CaPful of mechanisms regulating the mitochondrial permeability transitionFabio Di Lisa, Paolo BernardiThe Journal of Biological Chemistry|November 28, 2020
The energetic cost of NNT-dependent ROS removalNina Kaludercic, Fabio Di LisaPageof 64