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Scientific Reports|June 10, 2024
ProteinCoLoc streamlines Bayesian analysis of colocalization in microscopic imagesManuel Seefelder, Stefan Kochanek, Fabrice A C KleinJournal of Huntington'S Disease|July 24, 2022
Huntingtin and Its Partner Huntingtin-Associated Protein 40: Structural and Functional Considerations in Health and DiseaseManuel Seefelder, Fabrice A C Klein, Bernhard Landwehrmeyer, et al.The Journal of Biological Chemistry|November 16, 2004
Biochemical and NMR mapping of the interface between CREB-binding protein and ligand binding domains of nuclear receptor: beyond the LXXLL motifFabrice A C Klein, R Andrew Atkinson, Noelle Potier, et al.Human Molecular Genetics|April 27, 2011
Huntingtin affinity for partners is not changed by polyglutamine length: aggregation itself triggers aberrant interactionsAurélien Davranche, Hubert Aviolat, Gabrielle Zeder-Lutz, et al.Biochimica Et Biophysica Acta|June 1, 2016
Mammalian ataxin-2 modulates translation control at the pre-initiation complex via PI3K/mTOR and is induced by starvationIsabel Lastres-Becker, David Nonis, Florian Eich, et al.Human Molecular Genetics|June 20, 2013
Linear and extended: a common polyglutamine conformation recognized by the three antibodies MW1, 1C2 and 3B5H10Fabrice A C Klein, Gabrielle Zeder-Lutz, Alexandra Cousido-Siah, et al.Journal of Molecular Biology|June 15, 2007
Pathogenic and non-pathogenic polyglutamine tracts have similar structural properties: towards a length-dependent toxicity gradientFabrice A C Klein, Annalisa Pastore, Laura Masino, et al.Journal of Molecular Biology|September 30, 2018
SynAggreg: A Multifunctional High-Throughput Technology for Precision Study of Amyloid Aggregation and Systematic Discovery of Synergistic Inhibitor CompoundsHubert Aviolat, Yves Nominé, Sophie Gioria, et al.Journal of Molecular Biology|April 12, 2015
Structure of a single-chain Fv bound to the 17 N-terminal residues of huntingtin provides insights into pathogenic amyloid formation and suppressionErwin De Genst, Dimitri Y Chirgadze, Fabrice A C Klein, et al.Brain : a Journal of Neurology|December 27, 2013
The tumour suppressor gene WWOX is mutated in autosomal recessive cerebellar ataxia with epilepsy and mental retardationMartial Mallaret, Matthis Synofzik, Jaeho Lee, et al.Pageof 1