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Frontiers in Cellular Neuroscience|January 31, 2020
Mild Inactivation of RE-1 Silencing Transcription Factor (REST) Reduces Susceptibility to Kainic Acid-Induced SeizuresEmanuele Carminati, Federica Buffolo, Anna Rocchi, et al.Journal of Experimental Neuroscience|June 6, 2017
Optogenetic Modulation of Intracellular Signalling and Transcription: Focus on Neuronal PlasticityCyril Eleftheriou, Fabrizia Cesca, Luca Maragliano, et al.ACS Synthetic Biology|July 18, 2020
Engineering REST-Specific Synthetic PUF Proteins to Control Neuronal Gene Expression: A Combined Experimental and Computational StudyStefania Criscuolo, Mahad Gatti Iou, Assunta Merolla, et al.Frontiers in Cellular Neuroscience|November 30, 2023
Conditional knockout of REST/NRSF in excitatory neurons reduces seizure susceptibility to chemical kindlingGiulia Natali, Caterina Michetti, Alicja Krawczun-Rygmaczewska, et al.The Journal of Biological Chemistry|June 4, 2015
Functional Interaction between the Scaffold Protein Kidins220/ARMS and Neuronal Voltage-Gated Na+ ChannelsFabrizia Cesca, Annyesha Satapathy, Enrico Ferrea, et al.Molecular Biology of the Cell|November 3, 2006
Kidins220/ARMS is transported by a kinesin-1-based mechanism likely to be involved in neuronal differentiationAurora Bracale, Fabrizia Cesca, Veronika E Neubrand, et al.Journal of Cell Science|March 4, 2017
Neuronal hyperactivity causes Na+/H+ exchanger-induced extracellular acidification at active synapsesMartina Chiacchiaretta, Shahrzad Latifi, Mattia Bramini, et al.Frontiers in Neuroengineering|May 1, 2014
Bio-inspired hybrid microelectrodes: a hybrid solution to improve long-term performance of chronic intracortical implantsSara De Faveri, Emma Maggiolini, Ermanno Miele, et al.International Journal of Molecular Sciences|February 24, 2024
Alterations in KIDINS220/ARMS Expression Impact Sensory Processing and Social Behavior in Adult MiceMartina Albini, Amanda Almacellas-Barbanoj, Alicja Krawczun-Rygmaczewska, et al.Cerebral Cortex (New York, N.Y. : 1991)|February 28, 2012
Synaptic and extrasynaptic origin of the excitation/inhibition imbalance in the hippocampus of synapsin I/II/III knockout micePasqualina Farisello, Davide Boido, Thierry Nieus, et al.Pageof 6