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Fabrizio Pin

Showing results (41-50 of 56) with videos related to

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Elife|April 25, 2024
Deletion of FNDC5/irisin modifies murine osteocyte function in a sex-specific mannerAnika Shimonty, Fabrizio Pin, Matthew Prideaux, et al.
The American Journal of Pathology|February 12, 2013
Autophagic degradation contributes to muscle wasting in cancer cachexiaFabio Penna, Domiziana Costamagna, Fabrizio Pin, et al.
Biorxiv : the Preprint Server for Biology|November 21, 2023
Deletion of FNDC5/Irisin modifies murine osteocyte function in a sex-specific mannerAnika Shimonty, Fabrizio Pin, Matt Prideaux, et al.
Journal of Cachexia, Sarcopenia and Muscle|November 14, 2017
The mitochondrial metabolic reprogramming agent trimetazidine as an 'exercise mimetic' in cachectic C26-bearing miceFrancesca Molinari, Fabrizio Pin, Stefania Gorini, et al.
Scientific Reports|July 28, 2016
Autophagy is induced in the skeletal muscle of cachectic cancer patientsZaira Aversa, Fabrizio Pin, Simone Lucia, et al.
Scientific Reports|November 2, 2017
ACVR2B/Fc counteracts chemotherapy-induced loss of muscle and bone massRafael Barreto, Yukiko Kitase, Tsutomu Matsumoto, et al.
Journal of Cachexia, Sarcopenia and Muscle|July 17, 2018
Growth of ovarian cancer xenografts causes loss of muscle and bone mass: a new model for the study of cancer cachexiaFabrizio Pin, Rafael Barreto, Yukiko Kitase, et al.
Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|May 16, 2022
Triggering Receptor Expressed on Myeloid Cells 2 (TREM2) R47H Variant Causes Distinct Age- and Sex-Dependent Musculoskeletal Alterations in MiceAlyson L Essex, Joshua R Huot, Padmini Deosthale, et al.
Cancers|March 6, 2021
Targeting Mitochondria by SS-31 Ameliorates the Whole Body Energy Status in Cancer- and Chemotherapy-Induced CachexiaRiccardo Ballarò, Patrizia Lopalco, Valentina Audrito, et al.
Iscience|November 17, 2022
PGC-1α in the myofibers regulates the balance between myogenic and adipogenic progenitors affecting muscle regenerationMarc Beltrà, Fabrizio Pin, Domiziana Costamagna, et al.
Pageof 6

Showing results (41-50 of 56) with videos related to

Sort By:
Pageof 6
Elife|April 25, 2024
Deletion of FNDC5/irisin modifies murine osteocyte function in a sex-specific mannerAnika Shimonty, Fabrizio Pin, Matthew Prideaux, et al.
The American Journal of Pathology|February 12, 2013
Autophagic degradation contributes to muscle wasting in cancer cachexiaFabio Penna, Domiziana Costamagna, Fabrizio Pin, et al.
Biorxiv : the Preprint Server for Biology|November 21, 2023
Deletion of FNDC5/Irisin modifies murine osteocyte function in a sex-specific mannerAnika Shimonty, Fabrizio Pin, Matt Prideaux, et al.
Journal of Cachexia, Sarcopenia and Muscle|November 14, 2017
The mitochondrial metabolic reprogramming agent trimetazidine as an 'exercise mimetic' in cachectic C26-bearing miceFrancesca Molinari, Fabrizio Pin, Stefania Gorini, et al.
Scientific Reports|July 28, 2016
Autophagy is induced in the skeletal muscle of cachectic cancer patientsZaira Aversa, Fabrizio Pin, Simone Lucia, et al.
Scientific Reports|November 2, 2017
ACVR2B/Fc counteracts chemotherapy-induced loss of muscle and bone massRafael Barreto, Yukiko Kitase, Tsutomu Matsumoto, et al.
Journal of Cachexia, Sarcopenia and Muscle|July 17, 2018
Growth of ovarian cancer xenografts causes loss of muscle and bone mass: a new model for the study of cancer cachexiaFabrizio Pin, Rafael Barreto, Yukiko Kitase, et al.
Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|May 16, 2022
Triggering Receptor Expressed on Myeloid Cells 2 (TREM2) R47H Variant Causes Distinct Age- and Sex-Dependent Musculoskeletal Alterations in MiceAlyson L Essex, Joshua R Huot, Padmini Deosthale, et al.
Cancers|March 6, 2021
Targeting Mitochondria by SS-31 Ameliorates the Whole Body Energy Status in Cancer- and Chemotherapy-Induced CachexiaRiccardo Ballarò, Patrizia Lopalco, Valentina Audrito, et al.
Iscience|November 17, 2022
PGC-1α in the myofibers regulates the balance between myogenic and adipogenic progenitors affecting muscle regenerationMarc Beltrà, Fabrizio Pin, Domiziana Costamagna, et al.
Pageof 6