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Experimental Neurology|September 15, 2014
Increased neurotrophic factor levels in ventral mesencephalic cultures do not explain the protective effect of osteopontin and the synthetic 15-mer RGD domain against MPP+ toxicityLauren Broom, Peter Jenner, Sarah RoseTrends in Neurosciences|May 5, 2019
Dopaminergic Vulnerability in Parkinson Disease: The Cost of Humans' Habitual PerformanceLedia F Hernandez, Ignacio Obeso, Rui M Costa, et al.Parkinsonism & Related Disorders|December 22, 2020
Can adenosine A2A receptor antagonists be used to treat cognitive impairment, depression or excessive sleepiness in Parkinson's disease?Peter Jenner, Akihisa Mori, Tomoyuki KandaThe European Journal of Neuroscience|May 5, 2005
Increased osteopontin expression following intranigral lipopolysaccharide injection in the ratJoanna Iczkiewicz, Sarah Rose, Peter JennerBrain Research. Molecular Brain Research|November 19, 2004
Osteopontin (Eta-1) is present in the rat basal gangliaJoanna Iczkiewicz, Sarah Rose, Peter JennerExperimental Neurology|July 24, 2007
Osteopontin expression in activated glial cells following mechanical- or toxin-induced nigral dopaminergic cell lossJoanna Iczkiewicz, Sarah Rose, Peter JennerInternational Review of Neurobiology|September 23, 2023
How and why the adenosine A2A receptor became a target for Parkinson's disease therapyPeter Jenner, Tomoyuki Kanda, Akihisa MoriParkinsonism & Related Disorders|July 30, 2023
Importance of time to ON versus wearing OFF in total daily OFF time experienced by patients with Parkinson's diseaseStuart H Isaacson, Daniel Kremens, Yasar Torres-Yaghi, et al.Journal of Neurology|July 29, 2003
Dual dopamine agonist treatment in Parkinson's diseaseFabrizio Stocchi, Laura Vacca, Alfredo Berardelli, et al.Experimental Brain Research|March 26, 2013
Bilateral stimulation of the subthalamic nucleus has differential effects on reactive and proactive inhibition and conflict-induced slowing in Parkinson's diseaseIgnacio Obeso, Leonora Wilkinson, Maria-Cruz Rodríguez-Oroz, et al.Pageof 47