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Journal of Medical Genetics|June 26, 2012
Homozygous deletion of Tenascin-R in a patient with intellectual disabilityDavid Dufresne, Fadi F Hamdan, Jill A Rosenfeld, et al.Biochemistry|January 18, 2006
Use of an in situ disulfide cross-linking strategy to study the dynamic properties of the cytoplasmic end of transmembrane domain VI of the M3 muscarinic acetylcholine receptorStuart D C Ward, Fadi F Hamdan, Lanh M Bloodworth, et al.Biochemistry|February 6, 2008
Ligand-specific changes in M3 muscarinic acetylcholine receptor structure detected by a disulfide scanning strategyJian Hua Li, Fadi F Hamdan, Soo-Kyung Kim, et al.Journal of Medicinal Chemistry|February 27, 2007
Calcitonin gene-related peptide analogues with aza and indolizidinone amino acid residues reveal conformational requirements for antagonist activity at the human calcitonin gene-related peptide 1 receptorDamien Boeglin, Fadi F Hamdan, Rosa E Melendez, et al.The Journal of Biological Chemistry|August 12, 2005
Identification of an agonist-induced conformational change occurring adjacent to the ligand-binding pocket of the M(3) muscarinic acetylcholine receptorSung-Jun Han, Fadi F Hamdan, Soo-Kyung Kim, et al.Journal of Cell Science|April 26, 2007
Src-dependent phosphorylation of beta2-adaptin dissociates the beta-arrestin-AP-2 complexDelphine Fessart, May Simaan, Brandon Zimmerman, et al.Traffic (Copenhagen, Denmark)|January 11, 2014
CNIH4 interacts with newly synthesized GPCR and controls their export from the endoplasmic reticulumEtienne Sauvageau, Moulay D Rochdi, Morad Oueslati, et al.American Journal of Medical Genetics. Part A|July 9, 2026
Cerebellar Abnormalities in the Neuroimaging Spectrum of CLTC-Related DisorderDaniel Charouf, Andrea Accogli, Fadi F Hamdan, et al.The Journal of Biological Chemistry|August 7, 2007
Unraveling G protein-coupled receptor endocytosis pathways using real-time monitoring of agonist-promoted interaction between beta-arrestins and AP-2Fadi F Hamdan, Moulay Driss Rochdi, Billy Breton, et al.Human Mutation|April 14, 2015
A Gain-of-Function Mutation in NALCN in a Child with Intellectual Disability, Ataxia, and ArthrogryposisKyota Aoyagi, Elsa Rossignol, Fadi F Hamdan, et al.Pageof 10