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JCI Insight|July 18, 2024
Caspase-4/11 promotes hyperlipidemia and chronic kidney disease-accelerated vascular inflammation by enhancing trained immunityYu Sun, Yifan Lu, Lu Liu, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|May 28, 2020
Vascular Endothelial Cells and Innate ImmunityYing Shao, Jason Saredy, William Y Yang, et al.
Cells|June 10, 2023
Chronic Kidney Disease Transdifferentiates Veins into a Specialized Immune-Endocrine Organ with Increased MYCN-AP1 SignalingFatma Saaoud, Laisel Martinez, Yifan Lu, et al.
JCI Insight|October 8, 2021
IL-35 promotes CD4+Foxp3+ Tregs and inhibits atherosclerosis via maintaining CCR5-amplified Treg-suppressive mechanismsYing Shao, William Y Yang, Fatma Saaoud, et al.
Signal Transduction and Targeted Therapy|December 22, 2017
Central role of myeloid MCPIP1 in protecting against LPS-induced inflammation and lung injuryYong Li, Xuan Huang, Shengping Huang, et al.
Redox Biology|June 12, 2025
Uremic toxin receptor NR1H3 contributes to hyperlipidemia- and chronic kidney disease-accelerated vascular inflammation, which is partially suppressed by novel YBX2 anti-ROS pathwayYifan Lu, Yu Sun, Fatma Saaoud, et al.
Scientific Reports|December 13, 2016
Adiporedoxin suppresses endothelial activation via inhibiting MAPK and NF-κB signalingHui He, Fang Guo, Yong Li, et al.
Redox Biology|September 20, 2020
ROS systems are a new integrated network for sensing homeostasis and alarming stresses in organelle metabolic processesYu Sun, Yifan Lu, Jason Saredy, et al.
Redox Biology|June 28, 2020
Bone marrow deficiency of mRNA decaying protein Tristetraprolin increases inflammation and mitochondrial ROS but reduces hepatic lipoprotein production in LDLR knockout miceFatma Saaoud, Junfeng Wang, Stephen Iwanowycz, et al.
Frontiers in Immunology|July 12, 2021
Endothelial Immunity Trained by Coronavirus Infections, DAMP Stimulations and Regulated by Anti-Oxidant NRF2 May Contribute to Inflammations, Myelopoiesis, COVID-19 Cytokine Storms and ThromboembolismYing Shao, Jason Saredy, Keman Xu, et al.
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