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Cell Death and Differentiation|September 19, 2019
USP22 exerts tumor-suppressive functions in colorectal cancer by decreasing mTOR activityRobyn Laura Kosinsky, Maria Zerche, Dominik Saul, et al.
Oncotarget|October 3, 2015
Usp22 deficiency impairs intestinal epithelial lineage specification in vivoRobyn L Kosinsky, Florian Wegwitz, Nicole Hellbach, et al.
Cell Death and Differentiation|September 9, 2020
RNF40 exerts stage-dependent functions in differentiating osteoblasts and is essential for bone cell crosstalkZeynab Najafova, Peng Liu, Florian Wegwitz, et al.
Nucleic Acids Research|May 6, 2017
CHD1 regulates cell fate determination by activation of differentiation-induced genesSimon J Baumgart, Zeynab Najafova, Tareq Hossan, et al.
NPJ Breast Cancer|February 18, 2022
Estrogen receptor beta repurposes EZH2 to suppress oncogenic NFκB/p65 signaling in triple negative breast cancerKirsten G M Aspros, Jodi M Carter, Tanya L Hoskin, et al.
Oncogene|May 19, 2021
USP22 promotes HER2-driven mammary carcinoma aggressiveness by suppressing the unfolded protein responseEvangelos Prokakis, Anna Dyas, Regina Grün, et al.
Oncotarget|April 1, 2015
Overexpression of mutant Ptch in rhabdomyosarcomas is associated with promoter hypomethylation and increased Gli1 and H3K4me3 occupancyFrauke Nitzki, Ezequiel J Tolosa, Nicole Cuvelier, et al.
Hepatology (Baltimore, Md.)|April 15, 2026
Toxic lipid-induced epigenetic activation of ICAM1 in liver sinusoidal endothelium regulates myeloid-driven fibro-inflammatory response in MASHQianqian Guo, Nantawat Satthawiwat, Akitoshi Sano, et al.
Cell Communication and Signaling : CCS|February 12, 2024
USP22 supports the aggressive behavior of basal-like breast cancer by stimulating cellular respirationEvangelos Prokakis, Husam Bamahmoud, Shaishavi Jansari, et al.
American Journal of Physiology. Endocrinology and Metabolism|July 10, 2008
The glucocorticoid receptor and FOXO1 synergistically activate the skeletal muscle atrophy-associated MuRF1 geneDavid S Waddell, Leslie M Baehr, Jens van den Brandt, et al.
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