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Oncogene|January 13, 2018
p66Shc deficiency enhances CXCR4 and CCR7 recycling in CLL B cells by facilitating their dephosphorylation-dependent release from β-arrestin at early endosomesLaura Patrussi, Nagaja Capitani, Francesca Cattaneo, et al.
Plos One|July 7, 2012
HS1, a Lyn kinase substrate, is abnormally expressed in B-chronic lymphocytic leukemia and correlates with response to fludarabine-based regimenFederica Frezzato, Cristina Gattazzo, Veronica Martini, et al.
Frontiers in Cell and Developmental Biology|February 23, 2024
p66Shc deficiency in CLL cells enhances PD-L1 expression and suppresses immune synapse formationLudovica Lopresti, Nagaja Capitani, Vanessa Tatangelo, et al.
Cancers|March 28, 2024
Chronic Lymphocytic Leukemia (CLL) with Borderline Immunoglobulin Heavy Chain Mutational Status, a Rare Subgroup of CLL with Variable Disease CourseFrancesco Angotzi, Alessandro Cellini, Valeria Ruocco, et al.
Haematologica|June 17, 2017
Targeted activation of the SHP-1/PP2A signaling axis elicits apoptosis of chronic lymphocytic leukemia cellsElena Tibaldi, Mario Angelo Pagano, Federica Frezzato, et al.
Clinical Lymphoma, Myeloma & Leukemia|August 3, 2019
Prognostic and Predictive Effect of IGHV Mutational Status and Load in Chronic Lymphocytic Leukemia: Focus on FCR and BR TreatmentsAndrea Visentin, Monica Facco, Carmela Gurrieri, et al.
Oncotargets and Therapy|October 16, 2020
Lights and Shade of Next-Generation Pi3k Inhibitors in Chronic Lymphocytic LeukemiaAndrea Visentin, Federica Frezzato, Filippo Severin, et al.
Haematologica|September 7, 2014
Detection of monoclonal T populations in patients with KIR-restricted chronic lymphoproliferative disorder of NK cellsCristina Gattazzo, Antonella Teramo, Francesca Passeri, et al.
British Journal of Haematology|July 30, 2025
Serum soluble interleukin-2 receptor levels in hairy cell leukaemia as a marker of tumour burden with prognostic value and as a tool for disease monitoringFrancesco Angotzi, Alessandro Cellini, Nicolò Danesin, et al.
Blood|March 22, 2013
Intrinsic and extrinsic mechanisms contribute to maintain the JAK/STAT pathway aberrantly activated in T-type large granular lymphocyte leukemiaAntonella Teramo, Cristina Gattazzo, Francesca Passeri, et al.
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