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Free Radical Biology & Medicine|October 14, 2015
Scavenger Receptor B1 oxidative post-translational modifications are responsible for its loss in Rett syndromeGiuseppe Valacchi, Claudia Sticozzi, Giuseppe Belmonte, et al.Cell Death & Disease|July 20, 2020
The longevity-associated variant of BPIFB4 improves a CXCR4-mediated striatum-microglia crosstalk preventing disease progression in a mouse model of Huntington's diseaseAlba Di Pardo, Elena Ciaglia, Monica Cattaneo, et al.Molecular Therapy : the Journal of the American Society of Gene Therapy|September 18, 2022
Treatment with THI, an inhibitor of sphingosine-1-phosphate lyase, modulates glycosphingolipid metabolism and results therapeutically effective in experimental models of Huntington's diseaseGiuseppe Pepe, Luca Capocci, Federico Marracino, et al.Neurobiology of Disease|April 29, 2014
Oxidative brain damage in Mecp2-mutant murine models of Rett syndromeClaudio De Felice, Floriana Della Ragione, Cinzia Signorini, et al.Pageof 2