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EMBO Reports|August 5, 2006
Double-strand breaks arising by replication through a nick are repaired by cohesin-dependent sister-chromatid exchangeFelipe Cortés-Ledesma, Andrés AguileraNucleic Acids Research|October 2, 2007
Different genetic requirements for repair of replication-born double-strand breaks by sister-chromatid recombination and break-induced replicationFelipe Cortés-Ledesma, Cristina Tous, Andrés AguileraEMBO Reports|April 9, 2004
The absence of the yeast chromatin assembly factor Asf1 increases genomic instability and sister chromatid exchangeFélix Prado, Felipe Cortés-Ledesma, Andrés AguileraGenetics|September 30, 2004
A novel yeast mutation, rad52-L89F, causes a specific defect in Rad51-independent recombination that correlates with a reduced ability of Rad52-L89F to interact with Rad59Felipe Cortés-Ledesma, Francisco Malagón, Andrés AguileraFrontiers in Molecular Biosciences|January 31, 2020
"An End to a Means": How DNA-End Structure Shapes the Double-Strand Break Repair ProcessAlmudena Serrano-Benítez, Felipe Cortés-Ledesma, Jose F RuizCurrent Genetics|February 18, 2003
Mitotic recombination in Saccharomyces cerevisiaeFélix Prado, Felipe Cortés-Ledesma, Pablo Huertas, et al.Molecular Cell|June 25, 2003
Equal sister chromatid exchange is a major mechanism of double-strand break repair in yeastSergio González-Barrera, Felipe Cortés-Ledesma, Ralf E Wellinger, et al.The Journal of Biological Chemistry|October 30, 2010
TDP2/TTRAP is the major 5'-tyrosyl DNA phosphodiesterase activity in vertebrate cells and is critical for cellular resistance to topoisomerase II-induced DNA damageZhihong Zeng, Felipe Cortés-Ledesma, Sherif F El Khamisy, et al.Nature|August 22, 2008
CDK targets Sae2 to control DNA-end resection and homologous recombinationPablo Huertas, Felipe Cortés-Ledesma, Alessandro A Sartori, et al.Cell Cycle (Georgetown, Tex.)|April 4, 2007
SMC proteins, new players in the maintenance of genomic stabilityFelipe Cortés-Ledesma, Giaccomo de Piccoli, James E Haber, et al.Pageof 4