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Felix Wiedmann

Showing results (41-50 of 57) with videos related to

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Basic Research in Cardiology|June 9, 2018
Atrial fibrillation and heart failure-associated remodeling of two-pore-domain potassium (K<sub>2P</sub>) channels in murine disease models: focus on TASK-1Felix Wiedmann, Jan S Schulte, Bruna Gomes, et al.
Journal of the American Heart Association|January 22, 2026
<i>CAV1</i>-A Susceptibility Gene for Atrial Fibrillation: The Impact of Coding and Noncoding VariantsKristin Rädecke, David Rheinert, Annette Löwen, et al.
Heart Rhythm|December 12, 2022
Single beta-blocker or combined amiodarone therapy in implantable cardioverter-defibrillator and cardiac resynchronization therapy-defibrillator patients: Insights from the German DEVICE registryFelix Wiedmann, Hüseyin Ince, Christoph Stellbrink, et al.
Circulation. Genomic and Precision Medicine|December 21, 2018
Reduced Na<sup>+</sup> Current in Native Cardiomyocytes of a Brugada Syndrome Patient Associated With β-2-Syntrophin MutationConstanze Schmidt, Felix Wiedmann, Ibrahim El-Battrawy, et al.
Journal of the American Heart Association|May 12, 2020
Pharmacologic TWIK-Related Acid-Sensitive K+ Channel (TASK-1) Potassium Channel Inhibitor A293 Facilitates Acute Cardioversion of Paroxysmal Atrial Fibrillation in a Porcine Large Animal ModelFelix Wiedmann, Christoph Beyersdorf, Xiaobo Zhou, et al.
Progress in Biophysics and Molecular Biology|May 21, 2017
Stretch-activated two-pore-domain (K<sub>2P</sub>) potassium channels in the heart: Focus on atrial fibrillation and heart failureConstanze Schmidt, Felix Wiedmann, Stefan M Kallenberger, et al.
Frontiers in Physiology|October 24, 2018
Axial Tubule Junctions Activate Atrial Ca<sup>2+</sup> Release Across SpeciesSören Brandenburg, Jan Pawlowitz, Funsho E Fakuade, et al.
Communications Biology|January 8, 2025
A perfusion-independent high-throughput method to isolate liver sinusoidal endothelial cellsAnna Babin-Ebell Gonçalves, Yifang Mao, Tinja Baljkas, et al.
Basic Research in Cardiology|January 3, 2024
Acute antiarrhythmic effects of SGLT2 inhibitors-dapagliflozin lowers the excitability of atrial cardiomyocytesAmelie Paasche, Felix Wiedmann, Manuel Kraft, et al.
Circulation. Arrhythmia and Electrophysiology|September 14, 2019
Genetic Ablation of TASK-1 (Tandem of P Domains in a Weak Inward Rectifying K<sup>+</sup> Channel-Related Acid-Sensitive K<sup>+</sup> Channel-1) (K<sub>2P</sub>3.1) K<sup>+</sup> Channels Suppresses Atrial Fibrillation and Prevents Electrical RemodelingConstanze Schmidt, Felix Wiedmann, Christoph Beyersdorf, et al.
Pageof 6

Showing results (41-50 of 57) with videos related to

Sort By:
Pageof 6
Basic Research in Cardiology|June 9, 2018
Atrial fibrillation and heart failure-associated remodeling of two-pore-domain potassium (K<sub>2P</sub>) channels in murine disease models: focus on TASK-1Felix Wiedmann, Jan S Schulte, Bruna Gomes, et al.
Journal of the American Heart Association|January 22, 2026
<i>CAV1</i>-A Susceptibility Gene for Atrial Fibrillation: The Impact of Coding and Noncoding VariantsKristin Rädecke, David Rheinert, Annette Löwen, et al.
Heart Rhythm|December 12, 2022
Single beta-blocker or combined amiodarone therapy in implantable cardioverter-defibrillator and cardiac resynchronization therapy-defibrillator patients: Insights from the German DEVICE registryFelix Wiedmann, Hüseyin Ince, Christoph Stellbrink, et al.
Circulation. Genomic and Precision Medicine|December 21, 2018
Reduced Na<sup>+</sup> Current in Native Cardiomyocytes of a Brugada Syndrome Patient Associated With β-2-Syntrophin MutationConstanze Schmidt, Felix Wiedmann, Ibrahim El-Battrawy, et al.
Journal of the American Heart Association|May 12, 2020
Pharmacologic TWIK-Related Acid-Sensitive K+ Channel (TASK-1) Potassium Channel Inhibitor A293 Facilitates Acute Cardioversion of Paroxysmal Atrial Fibrillation in a Porcine Large Animal ModelFelix Wiedmann, Christoph Beyersdorf, Xiaobo Zhou, et al.
Progress in Biophysics and Molecular Biology|May 21, 2017
Stretch-activated two-pore-domain (K<sub>2P</sub>) potassium channels in the heart: Focus on atrial fibrillation and heart failureConstanze Schmidt, Felix Wiedmann, Stefan M Kallenberger, et al.
Frontiers in Physiology|October 24, 2018
Axial Tubule Junctions Activate Atrial Ca<sup>2+</sup> Release Across SpeciesSören Brandenburg, Jan Pawlowitz, Funsho E Fakuade, et al.
Communications Biology|January 8, 2025
A perfusion-independent high-throughput method to isolate liver sinusoidal endothelial cellsAnna Babin-Ebell Gonçalves, Yifang Mao, Tinja Baljkas, et al.
Basic Research in Cardiology|January 3, 2024
Acute antiarrhythmic effects of SGLT2 inhibitors-dapagliflozin lowers the excitability of atrial cardiomyocytesAmelie Paasche, Felix Wiedmann, Manuel Kraft, et al.
Circulation. Arrhythmia and Electrophysiology|September 14, 2019
Genetic Ablation of TASK-1 (Tandem of P Domains in a Weak Inward Rectifying K<sup>+</sup> Channel-Related Acid-Sensitive K<sup>+</sup> Channel-1) (K<sub>2P</sub>3.1) K<sup>+</sup> Channels Suppresses Atrial Fibrillation and Prevents Electrical RemodelingConstanze Schmidt, Felix Wiedmann, Christoph Beyersdorf, et al.
Pageof 6