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Felix Wiedmann

Showing results (51-60 of 57) with videos related to

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European Heart Journal|January 7, 2017
Inverse remodelling of K2P3.1 K+ channel expression and action potential duration in left ventricular dysfunction and atrial fibrillation: implications for patient-specific antiarrhythmic drug therapyConstanze Schmidt, Felix Wiedmann, Xiao-Bo Zhou, et al.
Circulation|May 9, 2015
Upregulation of K(2P)3.1 K+ Current Causes Action Potential Shortening in Patients With Chronic Atrial FibrillationConstanze Schmidt, Felix Wiedmann, Niels Voigt, et al.
Circulation|March 16, 2016
Response to Letter Regarding Article, "Upregulation of K2P3.1 K+ Current Causes Action Potential Shortening in Patients With Chronic Atrial Fibrillation"Constanze Schmidt, Felix Wiedmann, Niels Voigt, et al.
Cardiovascular Research|June 11, 2020
Altered atrial cytosolic calcium handling contributes to the development of postoperative atrial fibrillationFunsho E Fakuade, Vanessa Steckmeister, Fitzwilliam Seibertz, et al.
Cardiovascular Research|May 24, 2021
Treatment of atrial fibrillation with doxapram: TASK-1 potassium channel inhibition as a novel pharmacological strategyFelix Wiedmann, Christoph Beyersdorf, Xiao Bo Zhou, et al.
The Journal of Biological Chemistry|March 7, 2025
Rational design, synthesis, and evaluation of novel polypharmacological compounds targeting Na<sub>V</sub>1.5, K<sub>V</sub>1.5, and K<sub>2</sub>P channels for atrial fibrillationLorena Camargo-Ayala, Mauricio Bedoya, Albert Dasí, et al.
Circulation Research|October 15, 2025
Impaired Atrial Mitochondrial Calcium Handling in Patients With Atrial FibrillationJulius Ryan D Pronto, Fleur E Mason, Eva A Rog-Zielinska, et al.
Pageof 6

Showing results (51-60 of 57) with videos related to

Sort By:
Pageof 6
You have reached the last page of results.This site can display upto 57 results.
European Heart Journal|January 7, 2017
Inverse remodelling of K2P3.1 K+ channel expression and action potential duration in left ventricular dysfunction and atrial fibrillation: implications for patient-specific antiarrhythmic drug therapyConstanze Schmidt, Felix Wiedmann, Xiao-Bo Zhou, et al.
Circulation|May 9, 2015
Upregulation of K(2P)3.1 K+ Current Causes Action Potential Shortening in Patients With Chronic Atrial FibrillationConstanze Schmidt, Felix Wiedmann, Niels Voigt, et al.
Circulation|March 16, 2016
Response to Letter Regarding Article, "Upregulation of K2P3.1 K+ Current Causes Action Potential Shortening in Patients With Chronic Atrial Fibrillation"Constanze Schmidt, Felix Wiedmann, Niels Voigt, et al.
Cardiovascular Research|June 11, 2020
Altered atrial cytosolic calcium handling contributes to the development of postoperative atrial fibrillationFunsho E Fakuade, Vanessa Steckmeister, Fitzwilliam Seibertz, et al.
Cardiovascular Research|May 24, 2021
Treatment of atrial fibrillation with doxapram: TASK-1 potassium channel inhibition as a novel pharmacological strategyFelix Wiedmann, Christoph Beyersdorf, Xiao Bo Zhou, et al.
The Journal of Biological Chemistry|March 7, 2025
Rational design, synthesis, and evaluation of novel polypharmacological compounds targeting Na<sub>V</sub>1.5, K<sub>V</sub>1.5, and K<sub>2</sub>P channels for atrial fibrillationLorena Camargo-Ayala, Mauricio Bedoya, Albert Dasí, et al.
Circulation Research|October 15, 2025
Impaired Atrial Mitochondrial Calcium Handling in Patients With Atrial FibrillationJulius Ryan D Pronto, Fleur E Mason, Eva A Rog-Zielinska, et al.
Pageof 6