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Plos One|April 10, 2020
Proteomic changes during experimental de- and remyelination in the corpus callosumGabor T Szilagyi, Arkadiusz M Nawrocki, Krisztian Eros, et al.Antioxidants (Basel, Switzerland)|October 28, 2023
Highly Selective MIF Ketonase Inhibitor KRP-6 Diminishes M1 Macrophage Polarization and Metabolic ReprogrammingEszter Vámos, Nikoletta Kálmán, Eva Maria Sturm, et al.Journal of Molecular and Cellular Cardiology|May 24, 2006
PARP inhibition prevents postinfarction myocardial remodeling and heart failure via the protein kinase C/glycogen synthase kinase-3beta pathwayAnita Palfi, Ambrus Toth, Katalin Hanto, et al.Neurotoxicity Research|June 16, 2009
Protection against chronic hypoperfusion-induced retinal neurodegeneration by PARP inhibition via activation of PI-3-kinase Akt pathway and suppression of JNK and p38 MAP kinasesLaszlo Mester, Aliz Szabo, Tamas Atlasz, et al.Regulatory Peptides|September 2, 2006
The neuroprotective effects of PACAP in monosodium glutamate-induced retinal lesion involve inhibition of proapoptotic signaling pathwaysBoglárka Rácz, Ferenc Gallyas, Péter Kiss, et al.Antioxidants (Basel, Switzerland)|May 27, 2026
Selective MIF Enolase Inhibitor TE-91 Regulates M1 Polarization and Associated Metabolic ReprogrammingPéter Deák, Nikoletta Kálmán, Csenge Antus, et al.Journal of Molecular Neuroscience : MN|March 16, 2010
Effects of PACAP on mitochondrial apoptotic pathways and cytokine expression in rats subjected to renal ischemia/reperfusionGabriella Horvath, Boglarka Racz, Dora Reglodi, et al.Free Radical Biology & Medicine|August 10, 2006
A novel SOD-mimetic permeability transition inhibitor agent protects ischemic heart by inhibiting both apoptotic and necrotic cell deathZita Bognar, Tamas Kalai, Anita Palfi, et al.International Journal of Molecular Sciences|October 14, 2022
Remodeling of Liver and Plasma Lipidomes in Mice Lacking Cyclophilin DBalazs Koszegi, Gabor Balogh, Zoltan Berente, et al.Biochemical Pharmacology|January 30, 2018
Activation of mitochondrial fusion provides a new treatment for mitochondria-related diseasesAliz Szabo, Katalin Sumegi, Katalin Fekete, et al.Pageof 11