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Cells|June 19, 2024
Chronological and Biological Aging in Amyotrophic Lateral Sclerosis and the Potential of Senolytic TherapiesAnna Roshani Dashtmian, Fereshteh B Darvishi, William David ArnoldThe Journal of Neuroscience : the Official Journal of the Society for Neuroscience|November 12, 2025
The Neuromuscular Junction: A Shared Vulnerability in Aging and DiseaseKathryn R Moss, Fereshteh B Darvishi, Yomna Badawi, et al.Geroscience|January 17, 2025
5-HT2C agonism as a neurotherapeutic for sarcopenia: preclinical proof of conceptNathan R Kerr, Anna R Dashtmian, Fereshteh B Darvishi, et al.Experimental Physiology|March 28, 2025
Weighted cart pull: A novel outcome measure for sustained motor function in miceCharles D Brennan, Nathan R Kerr, Jose A Viteri, et al.Research Square|April 8, 2025
Targeting senescence in Amyotrophic Lateral Sclerosis: senolytic treatment improves neuromuscular function and preserves cortical excitability in a TDP-43Q331K mouse modelJose A Viteri, Nathan R Kerr, Charles D Brennan, et al.The Journal of Clinical Investigation|July 9, 2026
Neuromuscular junction failure in sarcopenia is linked to NaV1.4 loss and reversed by ClC-1 inhibitionW David Arnold, Jeanette Jeppesen Morgen, Pernille Bogetofte Thomasen, et al.Pageof 1