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Journal of the American Heart Association|August 6, 2014
Transgenic knockdown of cardiac sodium/glucose cotransporter 1 (SGLT1) attenuates PRKAG2 cardiomyopathy, whereas transgenic overexpression of cardiac SGLT1 causes pathologic hypertrophy and dysfunction in miceMohun Ramratnam, Ravi K Sharma, Stephen D'Auria, et al.Mayo Clinic Proceedings|February 7, 2021
COVID-19: Understanding Inter-Individual Variability and Implications for Precision MedicineNaveen L Pereira, Ferhaan Ahmad, Mirnela Byku, et al.Open Forum Infectious Diseases|November 7, 2018
<i>CFTR</i> Heterozygotes Are at Increased Risk of Respiratory Infections: A Population-Based StudyPhilip M Polgreen, Grant D Brown, Douglas B Hornick, et al.Journal of the American Heart Association|November 20, 2023
Validation of an Integrated Genetic-Epigenetic Test for the Assessment of Coronary Heart DiseaseRobert Philibert, Timur K Dogan, Stacey Knight, et al.Journal of Cardiac Surgery|October 15, 2011
Midterm outcomes of off-pump and on-pump coronary artery revascularization in renal transplant recipientsHossein Shayan, Rodolfo Rocha, Lawrence Wei, et al.Cell Cycle (Georgetown, Tex.)|November 26, 2008
E2F3 plays an essential role in cardiac development and functionJennifer C King, Ivan P G Moskowitz, Patrick G Burgon, et al.International Journal of Molecular Sciences|May 29, 2014
Rare variants in genes encoding MuRF1 and MuRF2 are modifiers of hypertrophic cardiomyopathyMing Su, Jizheng Wang, Lianming Kang, et al.Circulation|August 20, 2003
Consequences of pressure overload on sarcomere protein mutation-induced hypertrophic cardiomyopathyJoachim P Schmitt, Christopher Semsarian, Michael Arad, et al.Journal of Molecular and Cellular Cardiology|July 6, 2010
SGLT1, a novel cardiac glucose transporter, mediates increased glucose uptake in PRKAG2 cardiomyopathySanjay K Banerjee, David W Wang, Rodrigo Alzamora, et al.Molecular Medicine (Cambridge, Mass.)|December 28, 2012
High mobility group box 1 contributes to the pathogenesis of experimental pulmonary hypertension via activation of Toll-like receptor 4Eileen M Bauer, Richard Shapiro, Han Zheng, et al.Pageof 5