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The Journal of Clinical Investigation|March 6, 2003
IL-12 is required for differentiation of pathogenic CD8+ T cell effectors that cause myocarditisNir Grabie, Michael W Delfs, Jason R Westrich, et al.
Plos One|April 18, 2012
Early treatment with fumagillin, an inhibitor of methionine aminopeptidase-2, prevents Pulmonary Hypertension in monocrotaline-injured ratsDaniel J Kass, Eileen Rattigan, Rehan Kahloon, et al.
Plos One|July 23, 2014
Female sex is associated with worse prognosis in patients with hypertrophic cardiomyopathy in ChinaYilu Wang, Jizheng Wang, Yubao Zou, et al.
Circulation|January 14, 2009
Alterations of phospholamban function can exhibit cardiotoxic effects independent of excessive sarcoplasmic reticulum Ca2+-ATPase inhibitionJoachim P Schmitt, Ferhaan Ahmad, Kristina Lorenz, et al.
Circulation. Genomic and Precision Medicine|September 13, 2019
Heart Failure in the Era of Precision Medicine: A Scientific Statement From the American Heart AssociationSharon Cresci, Naveen L Pereira, Ferhaan Ahmad, et al.
Archives of Biochemistry and Biophysics|November 17, 2018
Pathogenic troponin T mutants with opposing effects on myofilament Ca<sup>2+</sup> sensitivity attenuate cardiomyopathy phenotypes in miceKarissa M Dieseldorff Jones, Yeojung Koh, Rebecca S Weller, et al.
Frontiers in Cardiovascular Medicine|April 27, 2026
Bioenergetic and metabolic aberrations in induced pluripotent stem cell-derived cardiomyocytes generated from a patient with Wolff-Parkinson-White syndrome caused a <i>PRKAG2</i> mutationPolina Baskin, Ifat Abramovich, Helena Milman, et al.
Science (New York, N.Y.)|March 1, 2003
Dilated cardiomyopathy and heart failure caused by a mutation in phospholambanJoachim P Schmitt, Mitsuhiro Kamisago, Michio Asahi, et al.
FEBS Letters|April 7, 2005
Functional role of AMP-activated protein kinase in the heart during exerciseNicolas Musi, Michael F Hirshman, Michael Arad, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 20, 2006
Cardiac myosin missense mutations cause dilated cardiomyopathy in mouse models and depress molecular motor functionJoachim P Schmitt, Edward P Debold, Ferhaan Ahmad, et al.
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