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Advances in Experimental Medicine and Biology|August 7, 2010
Role of the p53 family in stabilizing the genome and preventing polyploidizationFlaminia Talos, Ute M Moll
Molecular Cancer Research : MCR|April 12, 2011
Functional inactivation of endogenous MDM2 and CHIP by HSP90 causes aberrant stabilization of mutant p53 in human cancer cellsDun Li, Natalia D Marchenko, Ramona Schulz, et al.
Cancer Research|November 4, 2005
Mitochondrially targeted p53 has tumor suppressor activities in vivoFlaminia Talos, Oleksi Petrenko, Patricio Mena, et al.
Cell Cycle (Georgetown, Tex.)|June 28, 2008
E2F1 plays a direct role in Rb stabilization and p53-independent tumor suppressionGustavo Palacios, Flaminia Talos, Alice Nemajerova, et al.
Cancers|June 8, 2018
Gain-of-Function (GOF) Mutant p53 as Actionable Therapeutic TargetRamona Schulz-Heddergott, Ute M Moll
Molecular Cell|August 21, 2007
p73 suppresses polyploidy and aneuploidy in the absence of functional p53Flaminia Talos, Alice Nemajerova, Elsa R Flores, et al.
Oncoimmunology|December 18, 2012
HSP90 inhibitor antagonizing MIF: The specifics of pleiotropic cancer drug candidatesRamona Schulz, Matthias Dobbelstein, Ute M Moll
Cell Cycle (Georgetown, Tex.)|February 5, 2011
While p73 is essential, p63 is completely dispensable for the development of the central nervous systemLena Holembowski, Ramona Schulz, Flaminia Talos, et al.
The Journal of Comparative Neurology|February 7, 2014
Cortical hypoplasia and ventriculomegaly of p73-deficient mice: Developmental and adult analysisCarolina Medina-Bolívar, Emilio González-Arnay, Flaminia Talos, et al.
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