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Frédéric Checler

Showing results (1-10 of 119) with videos related to

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Prion|March 29, 2012
Two-steps control of cellular prion physiology by the extracellular regulated kinase-1 (ERK1)Frédéric Checler
Nature Medicine|September 10, 2013
Alzheimer's and prion diseases: PDK1 at the crossroadsFrédéric Checler
Therapie|December 15, 2010
[Production and catabolism of amyloid peptides in Alzheimer's disease]Frédéric Checler
The Journal of Biological Chemistry|April 10, 2016
Presenilin 1 and Presenilin 2 Target γ-Secretase Complexes to Distinct Cellular CompartmentsXavier Meckler, Frédéric Checler
Neurobiology of Disease|September 7, 2014
Eph receptors: new players in Alzheimer's disease pathogenesisMoustapha Cissé, Frédéric Checler
Journal of Alzheimer'S Disease : JAD|January 14, 2014
Visualization of specific γ-secretase complexes using bimolecular fluorescence complementationXavier Meckler, Frédéric Checler
Journal of Neurochemistry|March 8, 2005
Combined pharmacological, mutational and cell biology approaches indicate that p53-dependent caspase 3 activation triggered by cellular prion is dependent on its endocytosisClaire Sunyach, Frédéric Checler
Cells|December 4, 2020
Alterations of the Endoplasmic Reticulum (ER) Calcium Signaling Molecular Components in Alzheimer's DiseaseMounia Chami, Frédéric Checler
Trends in Neurosciences|November 26, 2002
Alzheimer's and prion diseases: distinct pathologies, common proteolytic denominatorsFrédéric Checler, Bruno Vincent
Current Alzheimer Research|February 26, 2020
Targeting Post-Translational Remodeling of Ryanodine Receptor: A New Track for Alzheimer's Disease Therapy?Mounia Chami, Frédéric Checler
Pageof 12

Showing results (1-10 of 119) with videos related to

Sort By:
Pageof 12
Prion|March 29, 2012
Two-steps control of cellular prion physiology by the extracellular regulated kinase-1 (ERK1)Frédéric Checler
Nature Medicine|September 10, 2013
Alzheimer's and prion diseases: PDK1 at the crossroadsFrédéric Checler
Therapie|December 15, 2010
[Production and catabolism of amyloid peptides in Alzheimer's disease]Frédéric Checler
The Journal of Biological Chemistry|April 10, 2016
Presenilin 1 and Presenilin 2 Target γ-Secretase Complexes to Distinct Cellular CompartmentsXavier Meckler, Frédéric Checler
Neurobiology of Disease|September 7, 2014
Eph receptors: new players in Alzheimer's disease pathogenesisMoustapha Cissé, Frédéric Checler
Journal of Alzheimer'S Disease : JAD|January 14, 2014
Visualization of specific γ-secretase complexes using bimolecular fluorescence complementationXavier Meckler, Frédéric Checler
Journal of Neurochemistry|March 8, 2005
Combined pharmacological, mutational and cell biology approaches indicate that p53-dependent caspase 3 activation triggered by cellular prion is dependent on its endocytosisClaire Sunyach, Frédéric Checler
Cells|December 4, 2020
Alterations of the Endoplasmic Reticulum (ER) Calcium Signaling Molecular Components in Alzheimer's DiseaseMounia Chami, Frédéric Checler
Trends in Neurosciences|November 26, 2002
Alzheimer's and prion diseases: distinct pathologies, common proteolytic denominatorsFrédéric Checler, Bruno Vincent
Current Alzheimer Research|February 26, 2020
Targeting Post-Translational Remodeling of Ryanodine Receptor: A New Track for Alzheimer's Disease Therapy?Mounia Chami, Frédéric Checler
Pageof 12