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The Journal of Biological Chemistry|December 5, 2002
Phosphorylation of key serine residues is required for internalization of the complement 5a (C5a) anaphylatoxin receptor via a beta-arrestin, dynamin, and clathrin-dependent pathwayLaurence Braun, Thierry Christophe, François BoulayBiochimie|April 13, 2007
The N-formyl peptide receptors and the anaphylatoxin C5a receptors: an overviewMarie-Josèphe Rabiet, Emilie Huet, François BoulayThe Journal of Biological Chemistry|September 6, 2008
Complement component 5a receptor oligomerization and homologous receptor down-regulationMarie-Josèphe Rabiet, Emilie Huet, François BoulayEuropean Journal of Immunology|July 19, 2005
Human mitochondria-derived N-formylated peptides are novel agonists equally active on FPR and FPRL1, while Listeria monocytogenes-derived peptides preferentially activate FPRMarie-Josèphe Rabiet, Emilie Huet, François BoulayThe Biochemical Journal|February 26, 2003
Direct binding of a fragment of the Wiskott-Aldrich syndrome protein to the C-terminal end of the anaphylatoxin C5a receptorMarianne Tardif, Laurence Brouchon, Marie-Josèphe Rabiet, et al.Cellular Signalling|June 28, 2007
The role of beta-arrestins in the formyl peptide receptor-like 1 internalization and signalingEmilie Huet, François Boulay, Sophie Barral, et al.Blood|August 15, 2002
Inhibitory effects of a dominant-interfering form of the Rho-GTPase Cdc42 in the chemoattractant-elicited signaling pathways leading to NADPH oxidase activation in differentiated HL-60 cellsMarie-Josèphe Rabiet, Marianne Tardif, Laurence Braun, et al.The Journal of Biological Chemistry|May 6, 2011
N-formyl peptide receptor 3 (FPR3) departs from the homologous FPR2/ALX receptor with regard to the major processes governing chemoattractant receptor regulation, expression at the cell surface, and phosphorylationMarie-Josèphe Rabiet, Laurence Macari, Claes Dahlgren, et al.Journal of Leukocyte Biology|June 14, 2005
Neutrophil NADPH-oxidase activation by an annexin AI peptide is transduced by the formyl peptide receptor (FPR), whereas an inhibitory signal is generated independently of the FPR family receptorsJennie Karlsson, Huamei Fu, François Boulay, et al.Journal of Immunology (Baltimore, Md. : 1950)|October 20, 2007
A monocyte-specific peptide from herpes simplex virus type 2 glycoprotein G activates the NADPH-oxidase but not chemotaxis through a G-protein-coupled receptor distinct from the members of the formyl peptide receptor familyLars Bellner, Jennie Karlsson, Huamei Fu, et al.Pageof 2