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Frances M Ashcroft

Showing results (51-60 of 172) with videos related to

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Physiology & Behavior|March 4, 2014
A mutation causing increased KATP channel activity leads to reduced anxiety in miceCarolina Lahmann, Rebecca H Clark, Michaela Iberl, et al.
Nature Reviews. Endocrinology|May 8, 2020
New insights into K<sub>ATP</sub> channel gene mutations and neonatal diabetes mellitusTanadet Pipatpolkai, Samuel Usher, Phillip J Stansfeld, et al.
The Journal of Physiology|September 5, 2018
Role of the C-terminus of SUR in the differential regulation of β-cell and cardiac K<sub>ATP</sub> channels by MgADP and metabolismNatascia Vedovato, Olof Rorsman, Konstantin Hennis, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 19, 2003
ATP-dependent interaction of the cytosolic domains of the inwardly rectifying K+ channel Kir6.2 revealed by fluorescence resonance energy transferTakashi Tsuboi, Jonathan D Lippiat, Frances M Ashcroft, et al.
The Journal of Physiology|February 4, 2003
Analysis of the differential modulation of sulphonylurea block of beta-cell and cardiac ATP-sensitive K+ (K(ATP)) channels by Mg-nucleotidesFrank Reimann, Michael Dabrowski, Phillippa Jones, et al.
Plos Computational Biology|May 11, 2010
Asymmetric switching in a homodimeric ABC transporter: a simulation studyJussi Aittoniemi, Heidi de Wet, Frances M Ashcroft, et al.
The FEBS Journal|June 18, 2010
The ATPase activities of sulfonylurea receptor 2A and sulfonylurea receptor 2B are influenced by the C-terminal 42 amino acidsHeidi de Wet, Constantina Fotinou, Nawaz Amad, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 16, 2007
Electrical inhibition of identified anorexigenic POMC neurons by orexin/hypocretinXiaosong Ma, Lejla Zubcevic, Jens C Brüning, et al.
Diabetes|May 30, 2006
Functional effects of mutations at F35 in the NH2-terminus of Kir6.2 (KCNJ11), causing neonatal diabetes, and response to sulfonylurea therapyPeter Proks, Christophe Girard, Halvor Baevre, et al.
The EMBO Journal|June 18, 2005
Kir6.2 mutations causing neonatal diabetes provide new insights into Kir6.2-SUR1 interactionsPaolo Tammaro, Christophe Girard, Janne Molnes, et al.
Pageof 18

Showing results (51-60 of 172) with videos related to

Sort By:
Pageof 18
Physiology & Behavior|March 4, 2014
A mutation causing increased KATP channel activity leads to reduced anxiety in miceCarolina Lahmann, Rebecca H Clark, Michaela Iberl, et al.
Nature Reviews. Endocrinology|May 8, 2020
New insights into K<sub>ATP</sub> channel gene mutations and neonatal diabetes mellitusTanadet Pipatpolkai, Samuel Usher, Phillip J Stansfeld, et al.
The Journal of Physiology|September 5, 2018
Role of the C-terminus of SUR in the differential regulation of β-cell and cardiac K<sub>ATP</sub> channels by MgADP and metabolismNatascia Vedovato, Olof Rorsman, Konstantin Hennis, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 19, 2003
ATP-dependent interaction of the cytosolic domains of the inwardly rectifying K+ channel Kir6.2 revealed by fluorescence resonance energy transferTakashi Tsuboi, Jonathan D Lippiat, Frances M Ashcroft, et al.
The Journal of Physiology|February 4, 2003
Analysis of the differential modulation of sulphonylurea block of beta-cell and cardiac ATP-sensitive K+ (K(ATP)) channels by Mg-nucleotidesFrank Reimann, Michael Dabrowski, Phillippa Jones, et al.
Plos Computational Biology|May 11, 2010
Asymmetric switching in a homodimeric ABC transporter: a simulation studyJussi Aittoniemi, Heidi de Wet, Frances M Ashcroft, et al.
The FEBS Journal|June 18, 2010
The ATPase activities of sulfonylurea receptor 2A and sulfonylurea receptor 2B are influenced by the C-terminal 42 amino acidsHeidi de Wet, Constantina Fotinou, Nawaz Amad, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 16, 2007
Electrical inhibition of identified anorexigenic POMC neurons by orexin/hypocretinXiaosong Ma, Lejla Zubcevic, Jens C Brüning, et al.
Diabetes|May 30, 2006
Functional effects of mutations at F35 in the NH2-terminus of Kir6.2 (KCNJ11), causing neonatal diabetes, and response to sulfonylurea therapyPeter Proks, Christophe Girard, Halvor Baevre, et al.
The EMBO Journal|June 18, 2005
Kir6.2 mutations causing neonatal diabetes provide new insights into Kir6.2-SUR1 interactionsPaolo Tammaro, Christophe Girard, Janne Molnes, et al.
Pageof 18