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Frances M Ashcroft

Showing results (81-90 of 172) with videos related to

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Plos One|June 5, 2019
Low extracellular magnesium does not impair glucose-stimulated insulin secretionLisanne M M Gommers, Thomas G Hill, Frances M Ashcroft, et al.
Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences|November 8, 2008
Review. SUR1: a unique ATP-binding cassette protein that functions as an ion channel regulatorJussi Aittoniemi, Constantina Fotinou, Tim J Craig, et al.
Plos One|December 6, 2011
FTO is expressed in neurones throughout the brain and its expression is unaltered by fastingJames S McTaggart, Sheena Lee, Michaela Iberl, et al.
British Journal of Pharmacology|January 29, 2005
Kir6.2-dependent high-affinity repaglinide binding to beta-cell K(ATP) channelsAnn Maria K Hansen, John Bondo Hansen, Richard D Carr, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 8, 2004
Molecular basis of Kir6.2 mutations associated with neonatal diabetes or neonatal diabetes plus neurological featuresPeter Proks, Jennifer F Antcliff, Jon Lippiat, et al.
Biochimica Et Biophysica Acta. Molecular Basis of Disease|December 6, 2017
FTO demethylase activity is essential for normal bone growth and bone mineralization in miceGregor Sachse, Chris Church, Michelle Stewart, et al.
Biophysical Journal|September 21, 2004
Nucleotide-dependent conformational changes in HisP: molecular dynamics simulations of an ABC transporter nucleotide-binding domainJeff D Campbell, Sundeep Singh Deol, Frances M Ashcroft, et al.
Brain : a Journal of Neurology|May 15, 2024
KATP channel mutation disrupts hippocampal network activity and nocturnal gamma shiftsMarie-Elisabeth Burkart, Josephine Kurzke, Robert Jacobi, et al.
The Journal of Clinical Endocrinology and Metabolism|April 9, 2009
An in-frame deletion in Kir6.2 (KCNJ11) causing neonatal diabetes reveals a site of interaction between Kir6.2 and SUR1Tim J Craig, Kenju Shimomura, Reinhard W Holl, et al.
Wellcome Open Research|August 10, 2021
Phenotype of a transient neonatal diabetes point mutation (SUR1-R1183W) in miceGregor Sachse, Elizabeth Haythorne, Peter Proks, et al.
Pageof 18

Showing results (81-90 of 172) with videos related to

Sort By:
Pageof 18
Plos One|June 5, 2019
Low extracellular magnesium does not impair glucose-stimulated insulin secretionLisanne M M Gommers, Thomas G Hill, Frances M Ashcroft, et al.
Philosophical Transactions of the Royal Society of London. Series B, Biological Sciences|November 8, 2008
Review. SUR1: a unique ATP-binding cassette protein that functions as an ion channel regulatorJussi Aittoniemi, Constantina Fotinou, Tim J Craig, et al.
Plos One|December 6, 2011
FTO is expressed in neurones throughout the brain and its expression is unaltered by fastingJames S McTaggart, Sheena Lee, Michaela Iberl, et al.
British Journal of Pharmacology|January 29, 2005
Kir6.2-dependent high-affinity repaglinide binding to beta-cell K(ATP) channelsAnn Maria K Hansen, John Bondo Hansen, Richard D Carr, et al.
Proceedings of the National Academy of Sciences of the United States of America|December 8, 2004
Molecular basis of Kir6.2 mutations associated with neonatal diabetes or neonatal diabetes plus neurological featuresPeter Proks, Jennifer F Antcliff, Jon Lippiat, et al.
Biochimica Et Biophysica Acta. Molecular Basis of Disease|December 6, 2017
FTO demethylase activity is essential for normal bone growth and bone mineralization in miceGregor Sachse, Chris Church, Michelle Stewart, et al.
Biophysical Journal|September 21, 2004
Nucleotide-dependent conformational changes in HisP: molecular dynamics simulations of an ABC transporter nucleotide-binding domainJeff D Campbell, Sundeep Singh Deol, Frances M Ashcroft, et al.
Brain : a Journal of Neurology|May 15, 2024
KATP channel mutation disrupts hippocampal network activity and nocturnal gamma shiftsMarie-Elisabeth Burkart, Josephine Kurzke, Robert Jacobi, et al.
The Journal of Clinical Endocrinology and Metabolism|April 9, 2009
An in-frame deletion in Kir6.2 (KCNJ11) causing neonatal diabetes reveals a site of interaction between Kir6.2 and SUR1Tim J Craig, Kenju Shimomura, Reinhard W Holl, et al.
Wellcome Open Research|August 10, 2021
Phenotype of a transient neonatal diabetes point mutation (SUR1-R1183W) in miceGregor Sachse, Elizabeth Haythorne, Peter Proks, et al.
Pageof 18