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Biorxiv : the Preprint Server for Biology|June 12, 2025
Parkinson's Disease-vulnerable and -resilient dopamine neurons display opposite responses to excitatory inputLotfi C Hadjas, Grace J Kollman, Lexe Linderhof, et al.Neurobiology of Disease|May 19, 2020
Evidence for the spread of human-derived mutant huntingtin protein in mice and non-human primatesPhilippe Gosset, Alexander Maxan, Melanie Alpaugh, et al.Neurobiology of Disease|December 13, 2023
Reducing huntingtin by immunotherapy delays disease progression in a mouse model of Huntington diseaseStefan Bartl, Yuanyun Xie, Nalini Potluri, et al.The Journal of Experimental Medicine|June 24, 2020
Neuronal interleukin-1 receptors mediate pain in chronic inflammatory diseasesBenoit Mailhot, Marine Christin, Nicolas Tessandier, et al.Proceedings of the National Academy of Sciences of the United States of America|July 14, 2016
Lmx1a and Lmx1b regulate mitochondrial functions and survival of adult midbrain dopaminergic neuronsHélène Doucet-Beaupré, Catherine Gilbert, Marcos Schaan Profes, et al.Human Molecular Genetics|September 11, 2014
Tau hyperphosphorylation and deregulation of calcineurin in mouse models of Huntington's diseaseMaud Gratuze, Anastasia Noël, Carl Julien, et al.Neurobiology of Disease|May 15, 2020
Inhibiting cellular uptake of mutant huntingtin using a monoclonal antibody: Implications for the treatment of Huntington's diseaseStefan Bartl, Abid Oueslati, Amber L Southwell, et al.Journal of Neurology|March 6, 2025
Tau levels in platelets isolated from Huntington's disease patients serve as a biomarker of disease severityMelanie Alpaugh, Juan Lantero-Rodriguez, Andrea L Benedet, et al.Acta Neuropathologica|May 26, 2016
Human-to-mouse prion-like propagation of mutant huntingtin proteinIksoo Jeon, Francesca Cicchetti, Giulia Cisbani, et al.Journal of Neurology, Neurosurgery, and Psychiatry|December 21, 2018
Platelet abnormalities in Huntington's diseaseHélèna L Denis, Jérôme Lamontagne-Proulx, Isabelle St-Amour, et al.Pageof 9