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Proceedings of the National Academy of Sciences of the United States of America|February 14, 2018
hMENA is a key regulator in endothelin-1/β-arrestin1-induced invadopodial function and metastatic processFrancesca Di Modugno, Valentina Caprara, Lidia Chellini, et al.Clinical & Experimental Metastasis|November 6, 2008
Identification of invasion specific splice variants of the cytoskeletal protein Mena present in mammary tumor cells during invasion in vivoSumanta Goswami, Ulrike Philippar, Daqian Sun, et al.Proceedings of the National Academy of Sciences of the United States of America|November 7, 2012
Splicing program of human MENA produces a previously undescribed isoform associated with invasive, mesenchymal-like breast tumorsFrancesca Di Modugno, Pierluigi Iapicca, Aaron Boudreau, et al.Cancer Research|March 17, 2007
Molecular cloning of hMena (ENAH) and its splice variant hMena+11a: epidermal growth factor increases their expression and stimulates hMena+11a phosphorylation in breast cancer cell linesFrancesca Di Modugno, Lucia DeMonte, Michele Balsamo, et al.Current Drug Targets|March 31, 2012
Molecular and genetic bases of pancreatic cancerVanja Vaccaro, Alain Gelibter, Emilio Bria, et al.Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|March 15, 2006
The cytoskeleton regulatory protein hMena (ENAH) is overexpressed in human benign breast lesions with high risk of transformation and human epidermal growth factor receptor-2-positive/hormonal receptor-negative tumorsFrancesca Di Modugno, Marcella Mottolese, Anna Di Benedetto, et al.Plos One|January 7, 2011
The cooperation between hMena overexpression and HER2 signalling in breast cancerFrancesca Di Modugno, Marcella Mottolese, Lucia DeMonte, et al.Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|August 5, 2008
Human Mena+11a isoform serves as a marker of epithelial phenotype and sensitivity to epidermal growth factor receptor inhibition in human pancreatic cancer cell linesMaria S Pino, Michele Balsamo, Francesca Di Modugno, et al.Journal for Immunotherapy of Cancer|January 27, 2026
Reciprocal regulation of hMENA and TGF-β signaling in cancer-associated fibroblasts promotes EMT, immunosuppression, poor prognosis, and ICT resistance in NSCLCRoberta Melchionna, Francesca Di Modugno, Anna Di Carlo, et al.International Journal of Cancer|March 18, 2004
Human Mena protein, a serex-defined antigen overexpressed in breast cancer eliciting both humoral and CD8+ T-cell immune responseFrancesca Di Modugno, Giovanna Bronzi, Matthew J Scanlan, et al.Pageof 3