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Francesca Longhena

Showing results (31-40 of 37) with videos related to

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Brain Pathology (Zurich, Switzerland)|January 14, 2018
Synapsin III is a key component of α-synuclein fibrils in Lewy bodies of PD brainsFrancesca Longhena, Gaia Faustini, Tatiana Varanita, et al.
Molecular Therapy : the Journal of the American Society of Gene Therapy|January 17, 2022
Synapsin III gene silencing redeems alpha-synuclein transgenic mice from Parkinson's disease-like phenotypeGaia Faustini, Francesca Longhena, Anna Masato, et al.
Laboratory Investigation; a Journal of Technical Methods and Pathology|March 31, 2015
Cavin-1 and Caveolin-1 are both required to support cell proliferation, migration and anchorage-independent cell growth in rhabdomyosarcomaFiorella Faggi, Nicola Chiarelli, Marina Colombi, et al.
Journal of Cell Science|May 14, 2015
α-synuclein and synapsin III cooperatively regulate synaptic function in dopamine neuronsMichela Zaltieri, Jessica Grigoletto, Francesca Longhena, et al.
Neurobiology of Disease|February 8, 2020
Alpha-synuclein/synapsin III pathological interplay boosts the motor response to methylphenidateGaia Faustini, Francesca Longhena, Agostino Bruno, et al.
Journal of Neuroinflammation|February 17, 2022
Dopamine signaling modulates microglial NLRP3 inflammasome activation: implications for Parkinson's diseaseAdrianne F Pike, Francesca Longhena, Gaia Faustini, et al.
Glia|October 12, 2020
Extracellular clusterin limits the uptake of α-synuclein fibrils by murine and human astrocytesAlice Filippini, Veronica Mutti, Gaia Faustini, et al.
Pageof 4

Showing results (31-40 of 37) with videos related to

Sort By:
Pageof 4
You have reached the last page of results.This site can display upto 37 results.
Brain Pathology (Zurich, Switzerland)|January 14, 2018
Synapsin III is a key component of α-synuclein fibrils in Lewy bodies of PD brainsFrancesca Longhena, Gaia Faustini, Tatiana Varanita, et al.
Molecular Therapy : the Journal of the American Society of Gene Therapy|January 17, 2022
Synapsin III gene silencing redeems alpha-synuclein transgenic mice from Parkinson's disease-like phenotypeGaia Faustini, Francesca Longhena, Anna Masato, et al.
Laboratory Investigation; a Journal of Technical Methods and Pathology|March 31, 2015
Cavin-1 and Caveolin-1 are both required to support cell proliferation, migration and anchorage-independent cell growth in rhabdomyosarcomaFiorella Faggi, Nicola Chiarelli, Marina Colombi, et al.
Journal of Cell Science|May 14, 2015
α-synuclein and synapsin III cooperatively regulate synaptic function in dopamine neuronsMichela Zaltieri, Jessica Grigoletto, Francesca Longhena, et al.
Neurobiology of Disease|February 8, 2020
Alpha-synuclein/synapsin III pathological interplay boosts the motor response to methylphenidateGaia Faustini, Francesca Longhena, Agostino Bruno, et al.
Journal of Neuroinflammation|February 17, 2022
Dopamine signaling modulates microglial NLRP3 inflammasome activation: implications for Parkinson's diseaseAdrianne F Pike, Francesca Longhena, Gaia Faustini, et al.
Glia|October 12, 2020
Extracellular clusterin limits the uptake of α-synuclein fibrils by murine and human astrocytesAlice Filippini, Veronica Mutti, Gaia Faustini, et al.
Pageof 4