Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Francesca Riuzzi

Showing results (1-10 of 46) with videos related to

Pageof 5
Sort By:
European Journal of Translational Myology|April 23, 2024
Report and Abstracts of the 20th Meeting of IIM, the Interuniversity Institute of Myology: Assisi, October 12-15, 2023Francesca Riuzzi, Emanuele Mocciaro
Journal of Cell Science|June 23, 2011
S100B protein regulates myoblast proliferation and differentiation by activating FGFR1 in a bFGF-dependent mannerFrancesca Riuzzi, Guglielmo Sorci, Rosario Donato
The American Journal of Pathology|July 21, 2007
RAGE expression in rhabdomyosarcoma cells results in myogenic differentiation and reduced proliferation, migration, invasiveness, and tumor growthFrancesca Riuzzi, Guglielmo Sorci, Rosario Donato
The Journal of Biological Chemistry|January 13, 2006
The amphoterin (HMGB1)/receptor for advanced glycation end products (RAGE) pair modulates myoblast proliferation, apoptosis, adhesiveness, migration, and invasiveness. Functional inactivation of RAGE in L6 myoblasts results in tumor formation in vivoFrancesca Riuzzi, Guglielmo Sorci, Rosario Donato
Journal of Cellular Physiology|January 19, 2006
S100B stimulates myoblast proliferation and inhibits myoblast differentiation by independently stimulating ERK1/2 and inhibiting p38 MAPKFrancesca Riuzzi, Guglielmo Sorci, Rosario Donato
Plos One|January 26, 2012
S100B engages RAGE or bFGF/FGFR1 in myoblasts depending on its own concentration and myoblast density. Implications for muscle regenerationFrancesca Riuzzi, Guglielmo Sorci, Sara Beccafico, et al.
Biochimica Et Biophysica Acta|October 30, 2012
RAGE in tissue homeostasis, repair and regenerationGuglielmo Sorci, Francesca Riuzzi, Ileana Giambanco, et al.
Journal of Cell Science|February 14, 2012
HMGB1-RAGE regulates muscle satellite cell homeostasis through p38-MAPK- and myogenin-dependent repression of Pax7 transcriptionFrancesca Riuzzi, Guglielmo Sorci, Roberta Sagheddu, et al.
Carcinogenesis|August 16, 2014
Defective RAGE activity in embryonal rhabdomyosarcoma cells results in high PAX7 levels that sustain migration and invasivenessSara Chiappalupi, Francesca Riuzzi, Stefania Fulle, et al.
Biomolecules|July 2, 2021
Hyperactivated RAGE in Comorbidities as a Risk Factor for Severe COVID-19-The Role of RAGE-RAS CrosstalkSara Chiappalupi, Laura Salvadori, Rosario Donato, et al.
Pageof 5

Showing results (1-10 of 46) with videos related to

Sort By:
Pageof 5
European Journal of Translational Myology|April 23, 2024
Report and Abstracts of the 20th Meeting of IIM, the Interuniversity Institute of Myology: Assisi, October 12-15, 2023Francesca Riuzzi, Emanuele Mocciaro
Journal of Cell Science|June 23, 2011
S100B protein regulates myoblast proliferation and differentiation by activating FGFR1 in a bFGF-dependent mannerFrancesca Riuzzi, Guglielmo Sorci, Rosario Donato
The American Journal of Pathology|July 21, 2007
RAGE expression in rhabdomyosarcoma cells results in myogenic differentiation and reduced proliferation, migration, invasiveness, and tumor growthFrancesca Riuzzi, Guglielmo Sorci, Rosario Donato
The Journal of Biological Chemistry|January 13, 2006
The amphoterin (HMGB1)/receptor for advanced glycation end products (RAGE) pair modulates myoblast proliferation, apoptosis, adhesiveness, migration, and invasiveness. Functional inactivation of RAGE in L6 myoblasts results in tumor formation in vivoFrancesca Riuzzi, Guglielmo Sorci, Rosario Donato
Journal of Cellular Physiology|January 19, 2006
S100B stimulates myoblast proliferation and inhibits myoblast differentiation by independently stimulating ERK1/2 and inhibiting p38 MAPKFrancesca Riuzzi, Guglielmo Sorci, Rosario Donato
Plos One|January 26, 2012
S100B engages RAGE or bFGF/FGFR1 in myoblasts depending on its own concentration and myoblast density. Implications for muscle regenerationFrancesca Riuzzi, Guglielmo Sorci, Sara Beccafico, et al.
Biochimica Et Biophysica Acta|October 30, 2012
RAGE in tissue homeostasis, repair and regenerationGuglielmo Sorci, Francesca Riuzzi, Ileana Giambanco, et al.
Journal of Cell Science|February 14, 2012
HMGB1-RAGE regulates muscle satellite cell homeostasis through p38-MAPK- and myogenin-dependent repression of Pax7 transcriptionFrancesca Riuzzi, Guglielmo Sorci, Roberta Sagheddu, et al.
Carcinogenesis|August 16, 2014
Defective RAGE activity in embryonal rhabdomyosarcoma cells results in high PAX7 levels that sustain migration and invasivenessSara Chiappalupi, Francesca Riuzzi, Stefania Fulle, et al.
Biomolecules|July 2, 2021
Hyperactivated RAGE in Comorbidities as a Risk Factor for Severe COVID-19-The Role of RAGE-RAS CrosstalkSara Chiappalupi, Laura Salvadori, Rosario Donato, et al.
Pageof 5