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Proceedings of the National Academy of Sciences of the United States of America|January 30, 2013
IKs channels open slowly because KCNE1 accessory subunits slow the movement of S4 voltage sensors in KCNQ1 pore-forming subunitsKatarina J Ruscic, Francesco Miceli, Carlos A Villalba-Galea, et al.
Frontiers in Pharmacology|June 21, 2011
The Voltage-Sensing Domain of K(v)7.2 Channels as a Molecular Target for Epilepsy-Causing Mutations and AnticonvulsantsFrancesco Miceli, Maria Virginia Soldovieri, Fabio Arturo Iannotti, et al.
The International Journal of Biochemistry & Cell Biology|February 7, 2006
Human neoplastic mesothelial cells express voltage-gated sodium channels involved in cell motilityGianluca Fulgenzi, Laura Graciotti, Monica Faronato, et al.
Epilepsia|November 15, 2021
Distinct epilepsy phenotypes and response to drugs in KCNA1 gain- and loss-of function variantsFrancesco Miceli, Renzo Guerrini, Mario Nappi, et al.
American Journal of Physiology. Cell Physiology|January 29, 2024
Kv7 channel activation reduces brain endothelial cell permeability and prevents kainic acid-induced blood-brain barrier damageCamilla Celentano, Lidia Carotenuto, Francesco Miceli, et al.
Epilepsia|December 20, 2014
A novel KCNQ3 mutation in familial epilepsy with focal seizures and intellectual disabilityFrancesco Miceli, Pasquale Striano, Maria Virginia Soldovieri, et al.
International Journal of Molecular Sciences|July 13, 2019
Epileptic Encephalopathy In A Patient With A Novel Variant In The Kv7.2 S2 Transmembrane Segment: Clinical, Genetic, and Functional FeaturesMaria Virginia Soldovieri, Paolo Ambrosino, Ilaria Mosca, et al.
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