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Frank C Church

Showing results (31-40 of 50) with videos related to

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Blood|March 1, 2011
Causal relationship between hyperfibrinogenemia, thrombosis, and resistance to thrombolysis in miceKellie R Machlus, Jessica C Cardenas, Frank C Church, et al.
Thrombosis Research|November 5, 2002
Inhibition of a thrombin anion-binding exosite-2 mutant by the glycosaminoglycan-dependent serpins protein C inhibitor and heparin cofactor IIScott T Cooper, Alireza R Rezaie, Charles T Esmon, et al.
Brain Sciences|May 28, 2022
Role of SARS-CoV-2 in Modifying Neurodegenerative Processes in Parkinson's Disease: A Narrative ReviewJeremy M Morowitz, Kaylyn B Pogson, Daniel A Roque, et al.
Blood|August 26, 2010
Protease-activated receptors mediate crosstalk between coagulation and fibrinolysisTroy A McEachron, Rafal Pawlinski, Kristy L Richards, et al.
The FEBS Journal|September 24, 2005
Control of the coagulation system by serpins. Getting by with a little help from glycosaminoglycansRobert N Pike, Ashley M Buckle, Bernard F le Bonniec, et al.
Biochimica Et Biophysica Acta|June 24, 2003
Basic residues in the 37-loop of activated protein C modulate inhibition by protein C inhibitor but not by alpha(1)-antitrypsinLaura N Glasscock, Bruce Gerlitz, Scott T Cooper, et al.
Blood|July 2, 2002
Activated protein C cleaves factor Va more efficiently on endothelium than on platelet surfacesJulie A Oliver, Dougald M Monroe, Frank C Church, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 10, 2002
Crystal structures of native and thrombin-complexed heparin cofactor II reveal a multistep allosteric mechanismTrevor P Baglin, Robin W Carrell, Frank C Church, et al.
Cardiology|November 24, 2016
Protease-Activated Receptor 1 Contributes to Angiotensin II-Induced Cardiovascular Remodeling and InflammationSilvio Antoniak, Jessica C Cardenas, Laura J Buczek, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|January 15, 2011
Overexpression of the cell cycle inhibitor p16INK4a promotes a prothrombotic phenotype following vascular injury in miceJessica C Cardenas, A Phillip Owens, Janakiraman Krishnamurthy, et al.
Pageof 5

Showing results (31-40 of 50) with videos related to

Sort By:
Pageof 5
Blood|March 1, 2011
Causal relationship between hyperfibrinogenemia, thrombosis, and resistance to thrombolysis in miceKellie R Machlus, Jessica C Cardenas, Frank C Church, et al.
Thrombosis Research|November 5, 2002
Inhibition of a thrombin anion-binding exosite-2 mutant by the glycosaminoglycan-dependent serpins protein C inhibitor and heparin cofactor IIScott T Cooper, Alireza R Rezaie, Charles T Esmon, et al.
Brain Sciences|May 28, 2022
Role of SARS-CoV-2 in Modifying Neurodegenerative Processes in Parkinson's Disease: A Narrative ReviewJeremy M Morowitz, Kaylyn B Pogson, Daniel A Roque, et al.
Blood|August 26, 2010
Protease-activated receptors mediate crosstalk between coagulation and fibrinolysisTroy A McEachron, Rafal Pawlinski, Kristy L Richards, et al.
The FEBS Journal|September 24, 2005
Control of the coagulation system by serpins. Getting by with a little help from glycosaminoglycansRobert N Pike, Ashley M Buckle, Bernard F le Bonniec, et al.
Biochimica Et Biophysica Acta|June 24, 2003
Basic residues in the 37-loop of activated protein C modulate inhibition by protein C inhibitor but not by alpha(1)-antitrypsinLaura N Glasscock, Bruce Gerlitz, Scott T Cooper, et al.
Blood|July 2, 2002
Activated protein C cleaves factor Va more efficiently on endothelium than on platelet surfacesJulie A Oliver, Dougald M Monroe, Frank C Church, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 10, 2002
Crystal structures of native and thrombin-complexed heparin cofactor II reveal a multistep allosteric mechanismTrevor P Baglin, Robin W Carrell, Frank C Church, et al.
Cardiology|November 24, 2016
Protease-Activated Receptor 1 Contributes to Angiotensin II-Induced Cardiovascular Remodeling and InflammationSilvio Antoniak, Jessica C Cardenas, Laura J Buczek, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|January 15, 2011
Overexpression of the cell cycle inhibitor p16INK4a promotes a prothrombotic phenotype following vascular injury in miceJessica C Cardenas, A Phillip Owens, Janakiraman Krishnamurthy, et al.
Pageof 5