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Blood|January 16, 2007
TLR ligands differentially affect uptake and presentation of cellular antigensMarkus Michael Weck, Frank Grünebach, Daniela Werth, et al.Cancer Research|June 3, 2006
BCR-ABL is not an immunodominant antigen in chronic myelogenous leukemiaFrank Grünebach, Valbona Mirakaj, Valdete Mirakaj, et al.Experimental Hematology|June 14, 2002
Monoclonal antibody 9C4 recognizes epithelial cellular adhesion molecule, a cell surface antigen expressed in early steps of erythropoiesisReiner Lammers, Christina Giesert, Frank Grünebach, et al.Blood|August 18, 2005
PPAR-gamma agonists inhibit toll-like receptor-mediated activation of dendritic cells via the MAP kinase and NF-kappaB pathwaysSilke Appel, Valdete Mirakaj, Anita Bringmann, et al.Blood|December 29, 2005
Epithelial-specific transcription factor ESE-3 is involved in the development of monocyte-derived DCsSilke Appel, Anita Bringmann, Frank Grünebach, et al.Haematologica|February 27, 2003
Heterogeneity among human bone marrow-derived mesenchymal stem cells and neural progenitor cellsWichard Vogel, Frank Grünebach, Conrad A Messam, et al.Experimental Hematology|September 13, 2002
Cyclopentenone prostaglandins induce caspase activation and apoptosis in dendritic cells by a PPAR-gamma-independent mechanism: regulation by inflammatory and T cell-derived stimuliAlessio Nencioni, Kirsten Lauber, Frank Grünebach, et al.Cancer Gene Therapy|May 7, 2005
Cotransfection of dendritic cells with RNA coding for HER-2/neu and 4-1BBL increases the induction of tumor antigen specific cytotoxic T lymphocytesFrank Grünebach, Katrin Kayser, Markus M Weck, et al.Clinical Cancer Research : an Official Journal of the American Association for Cancer Research|July 4, 2007
Histone deacetylase inhibitors affect dendritic cell differentiation and immunogenicityAlessio Nencioni, Julia Beck, Daniela Werth, et al.Journal of Immunology (Baltimore, Md. : 1950)|November 11, 2003
Cyclopentenone prostaglandins induce lymphocyte apoptosis by activating the mitochondrial apoptosis pathway independent of external death receptor signalingAlessio Nencioni, Kirsten Lauber, Frank Grünebach, et al.Pageof 5