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Frank M Laferla

Showing results (11-20 of 212) with videos related to

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Current Alzheimer Research|April 10, 2009
M1 agonists as a potential disease-modifying therapy for Alzheimer's diseaseAntonella Caccamo, Abraham Fisher, Frank M LaFerla
Journal of Neurochemistry|February 3, 2009
Chronic copper exposure exacerbates both amyloid and tau pathology and selectively dysregulates cdk5 in a mouse model of ADMasashi Kitazawa, David Cheng, Frank M Laferla
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 8, 2003
Ca2+ signaling in mouse cortical neurons studied by two-photon imaging and photoreleased inositol triphosphateGrace E Stutzmann, Frank M LaFerla, Ian Parker
Current Alzheimer Research|June 25, 2005
Amyloid beta-peptide: the inside storyBertrand P Tseng, Masashi Kitazawa, Frank M LaFerla
Journal of Alzheimer'S Disease : JAD|May 29, 2012
Elucidating the triggers, progression, and effects of Alzheimer's diseaseRodrigo Medeiros, Meredith A Chabrier, Frank M LaFerla
Annals of Neurology|March 6, 2008
Inflammation induces tau pathology in inclusion body myositis model via glycogen synthase kinase-3betaMasashi Kitazawa, Dan N Trinh, Frank M LaFerla
Nature Reviews. Neuroscience|June 7, 2007
Intracellular amyloid-beta in Alzheimer's diseaseFrank M LaFerla, Kim N Green, Salvatore Oddo
Annals of the New York Academy of Sciences|February 1, 2005
Microglia as a potential bridge between the amyloid beta-peptide and tauMasashi Kitazawa, Tritia R Yamasaki, Frank M LaFerla
CNS Neuroscience & Therapeutics|June 18, 2010
The role of tau in Alzheimer's disease and related disordersRodrigo Medeiros, David Baglietto-Vargas, Frank M LaFerla
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|March 19, 2011
Presenilin is necessary for efficient proteolysis through the autophagy-lysosome system in a γ-secretase-independent mannerKara M Neely, Kim N Green, Frank M LaFerla
Pageof 22

Showing results (11-20 of 212) with videos related to

Sort By:
Pageof 22
Current Alzheimer Research|April 10, 2009
M1 agonists as a potential disease-modifying therapy for Alzheimer's diseaseAntonella Caccamo, Abraham Fisher, Frank M LaFerla
Journal of Neurochemistry|February 3, 2009
Chronic copper exposure exacerbates both amyloid and tau pathology and selectively dysregulates cdk5 in a mouse model of ADMasashi Kitazawa, David Cheng, Frank M Laferla
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 8, 2003
Ca2+ signaling in mouse cortical neurons studied by two-photon imaging and photoreleased inositol triphosphateGrace E Stutzmann, Frank M LaFerla, Ian Parker
Current Alzheimer Research|June 25, 2005
Amyloid beta-peptide: the inside storyBertrand P Tseng, Masashi Kitazawa, Frank M LaFerla
Journal of Alzheimer'S Disease : JAD|May 29, 2012
Elucidating the triggers, progression, and effects of Alzheimer's diseaseRodrigo Medeiros, Meredith A Chabrier, Frank M LaFerla
Annals of Neurology|March 6, 2008
Inflammation induces tau pathology in inclusion body myositis model via glycogen synthase kinase-3betaMasashi Kitazawa, Dan N Trinh, Frank M LaFerla
Nature Reviews. Neuroscience|June 7, 2007
Intracellular amyloid-beta in Alzheimer's diseaseFrank M LaFerla, Kim N Green, Salvatore Oddo
Annals of the New York Academy of Sciences|February 1, 2005
Microglia as a potential bridge between the amyloid beta-peptide and tauMasashi Kitazawa, Tritia R Yamasaki, Frank M LaFerla
CNS Neuroscience & Therapeutics|June 18, 2010
The role of tau in Alzheimer's disease and related disordersRodrigo Medeiros, David Baglietto-Vargas, Frank M LaFerla
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|March 19, 2011
Presenilin is necessary for efficient proteolysis through the autophagy-lysosome system in a γ-secretase-independent mannerKara M Neely, Kim N Green, Frank M LaFerla
Pageof 22